Loss of net as repressor leads to constitutive increased c-fos transcription in cervical cancer cells

被引:27
作者
van Riggelen, J
Buchwalter, G
Soto, U
Arce, JD
Hausen, HZ
Wasylyk, B
Rösl, F
机构
[1] Deutsch Krebsforschungszentrum, Forsch Schwerpunkt Angewandte Tumorvirol, D-69120 Heidelberg, Germany
[2] ULP, INSERM, CNRS, Inst Genet & Biol Mol & Cellulaire, F-67404 Illkirch Graffenstaden, France
[3] Loma Linda Univ, Sch Med, Dept Biochem, Loma Linda, CA 92350 USA
关键词
D O I
10.1074/jbc.M409915200
中图分类号
Q5 [生物化学]; Q7 [分子生物学];
学科分类号
071010 ; 081704 ;
摘要
We have investigated the expression of c-fos in cervical carcinoma cells and in somatic cell hybrids derived therefrom. In malignant cells, c-fos was constitutively expressed even after serum starvation. Dissection of the c-fos promoter showed that expression was mainly controlled by the SRE motif, which was active in malignant cells, but repressed in their non-malignant counterparts. Constitutive SRE activity was not mediated by sustained mitogen-activated protein kinase activity but because of inefficient expression of the ternary complex factor Net, which was either very low or even barely discernible. Chromatin immunoprecipitation assays revealed that Net directly binds to the SRE nucleoprotein complex in non-tumorigenic cells, but not in malignant segregants. Small interfering RNA targeted against Net resulted in enhanced c-fos transcription, clearly illustrating its repressor function. Conversely, stable ectopic expression of Net in malignant cells negatively regulated endogenous c-fos, resulting in a disappearance of the c-Fos protein from the AP-1 transcription complex. These data indicate that loss of Net and constitutive c-fos expression appear to be a key event in the transformation of cervical cancer cells.
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收藏
页码:3286 / 3294
页数:9
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