Modulating the immune response in demyelinating diseases

被引:4
作者
Hughes, R [1 ]
机构
[1] United Med & Dent Sch Guys & St Thomas Hosp, Guys Hosp, Dept Neurol, London SE1 9RT, England
关键词
D O I
10.1136/jnnp.64.2.148
中图分类号
R74 [神经病学与精神病学];
学科分类号
摘要
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页码:148 / 148
页数:1
相关论文
共 8 条
[1]   REGULATORY T-CELL CLONES INDUCED BY ORAL TOLERANCE - SUPPRESSION OF AUTOIMMUNE ENCEPHALOMYELITIS [J].
CHEN, YH ;
KUCHROO, VK ;
INOBE, J ;
HAFLER, DA ;
WEINER, HL .
SCIENCE, 1994, 265 (5176) :1237-1240
[2]  
FAVA RA, 1990, BLOOD, V76, P1946
[3]  
Kiefer R, 1996, AM J PATHOL, V148, P211
[4]  
OSSEGE LM, 1997, AKUTELLE NEUROLOGIE, V24, pS49
[5]  
Ruuls SR, 1996, J IMMUNOL, V157, P5721
[6]   ELEVATED SERUM LEVELS OF TUMOR-NECROSIS-FACTOR-ALPHA IN GUILLAIN-BARRE-SYNDROME [J].
SHARIEF, MK ;
MCLEAN, B ;
THOMPSON, EJ .
ANNALS OF NEUROLOGY, 1993, 33 (06) :591-596
[7]   Potential role of transforming growth factor-beta 1 in terminating the immune response in patients with Guillain-Barre syndrome [J].
Sindern, E ;
Schweppe, K ;
Ossege, LM ;
Malin, JP .
JOURNAL OF NEUROLOGY, 1996, 243 (03) :264-268
[8]   Dynamics of mRNA expression of interferon-gamma, interleukin 4 and transforming growth factor beta 1 in sciatic nerves and lymphoid organs In experimental allergic neuritis [J].
Zhu, J ;
Mix, E ;
Issazadeh, S ;
Link, H .
EUROPEAN JOURNAL OF NEUROLOGY, 1996, 3 (03) :232-240