Antibodies to DNA

[1] NETWORK THEORY IN AUTOIMMUNITY - INVITRO SUPPRESSION OF SERUM ANTI-DNA ANTIBODY-BINDING TO DNA BY ANTI-IDIOTYPIC ANTIBODY IN SYSTEMIC LUPUS-ERYTHEMATOSUS [J]. JOURNAL OF CLINICAL INVESTIGATION, 1981, 67 (05) : 1297 - 1304

[2] ALARCONSEGOVIA D, 1983, CLIN EXP IMMUNOL, V52, P365

[3] PREVENTION OF RELAPSES IN SYSTEMIC LUPUS-ERYTHEMATOSUS [J]. LANCET, 1995, 345 (8965) : 1595 - 1599

[4] ANTI-DNA ANTIBODIES AND LUPUS NEPHRITIS - THE COMPLEXITY OF CROSS-REACTIVITY [J]. IMMUNOLOGY TODAY, 1990, 11 (07): : 232 - 234

[5] THE CENTRAL ROLE OF CHROMATIN IN AUTOIMMUNE RESPONSES TO HISTONES AND DNA IN SYSTEMIC LUPUS-ERYTHEMATOSUS [J]. JOURNAL OF CLINICAL INVESTIGATION, 1994, 94 (01) : 184 - 192

[6] Ultraviolet Light-induced keratinocyte apoptosis: A potential mechanism for the induction of skin lesions and autoantibody production in LE [J]. LUPUS, 1997, 6 (02) : 175 - 180

[7] CEPPELLINI R, 1957, P SOC EXP BIOL MED, V96, P572

[8] DESAI DD, 1993, J IMMUNOL, V151, P1614

[9] THE ROLE OF SOMATIC MUTATION IN THE PATHOGENIC ANTI-DNA RESPONSE [J]. ANNUAL REVIEW OF IMMUNOLOGY, 1992, 10 : 731 - 757

[10] Skewed distribution of IgG Fc receptor IIa (CD32) polymorphism is associated with renal disease in systemic lupus erythematosus patients [J]. ARTHRITIS AND RHEUMATISM, 1995, 38 (12): : 1832 - 1836

[1] NETWORK THEORY IN AUTOIMMUNITY - INVITRO SUPPRESSION OF SERUM ANTI-DNA ANTIBODY-BINDING TO DNA BY ANTI-IDIOTYPIC ANTIBODY IN SYSTEMIC LUPUS-ERYTHEMATOSUS [J]. JOURNAL OF CLINICAL INVESTIGATION, 1981, 67 (05) : 1297 - 1304

[2] ALARCONSEGOVIA D, 1983, CLIN EXP IMMUNOL, V52, P365

[3] PREVENTION OF RELAPSES IN SYSTEMIC LUPUS-ERYTHEMATOSUS [J]. LANCET, 1995, 345 (8965) : 1595 - 1599

[4] ANTI-DNA ANTIBODIES AND LUPUS NEPHRITIS - THE COMPLEXITY OF CROSS-REACTIVITY [J]. IMMUNOLOGY TODAY, 1990, 11 (07): : 232 - 234

[5] THE CENTRAL ROLE OF CHROMATIN IN AUTOIMMUNE RESPONSES TO HISTONES AND DNA IN SYSTEMIC LUPUS-ERYTHEMATOSUS [J]. JOURNAL OF CLINICAL INVESTIGATION, 1994, 94 (01) : 184 - 192

[6] Ultraviolet Light-induced keratinocyte apoptosis: A potential mechanism for the induction of skin lesions and autoantibody production in LE [J]. LUPUS, 1997, 6 (02) : 175 - 180

[7] CEPPELLINI R, 1957, P SOC EXP BIOL MED, V96, P572

[8] DESAI DD, 1993, J IMMUNOL, V151, P1614

[9] THE ROLE OF SOMATIC MUTATION IN THE PATHOGENIC ANTI-DNA RESPONSE [J]. ANNUAL REVIEW OF IMMUNOLOGY, 1992, 10 : 731 - 757

[10] Skewed distribution of IgG Fc receptor IIa (CD32) polymorphism is associated with renal disease in systemic lupus erythematosus patients [J]. ARTHRITIS AND RHEUMATISM, 1995, 38 (12): : 1832 - 1836