Sevoflurane postconditioning prevents activation of caspase 3 and 9 through antiapoptotic signaling after myocardial ischemia–reperfusion

[1] Stadnicka A(2007)Volatile anesthetic-induced cardiac preconditioning J Anesth 21 212-219

[2] Marinovic J(2004)Mechanisms of cardioprotection by volatile anesthetics Anesthesiology 100 707-721

[3] Ljubkovic M(2004)Adenosine and a nitric oxide donor enhances cardioprotection by preconditioning with isoflurane through mitochondrial adenosine triphosphate-sensitive K+ channel-dependent and -independent mechanisms Anesthesiology 100 515-524

[4] Bienengraeber MW(2008)Sevoflurane enhances ethanol-induced cardiac preconditioning through modulation of protein kinase C, mitochondrial KATP channels, and nitric oxide synthase, in guinea pig hearts Anesth Analg 106 9-16

[5] Bosnjak ZJ(2009)Acute memory phase of sevoflurane preconditioning is associated with sustained translocation of protein kinase C-α and ε, but not δ, in isolated guinea pig hearts Eur J Anaesthesiol 26 582-43

[6] Tanaka K(2009)Aprotinin abolishes sevoflurane postconditioning by inhibiting nitric oxide production and phosphorylation of protein kinase C-δ and glycogen synthase kinase 3β Anesthesiology 111 1036-765

[7] Ludwig LM(2008)Postconditioning by volatile anesthetics: salvaging ischemic myocardium at reperfusion by activation of prosurvival signaling J Cardiothorac Vasc Anesth 22 753-90

[8] Kersten JR(2001)Progressively developed myocardial apoptotic cell death during late phase of reperfusion Apoptosis 6 279-79

[9] Pagel PS(2006)Immunohistochemical expression of activated caspase-3 in human myocardial infarction Virchows Arch 448 75-460

[10] Warltier DC(2004)New directions for protecting the heart against ischaemia-reperfusion injury: targeting the reperfusion injury salvage kinase (RISK)-pathway Cardiovasc Res 61 448-232

[1] Stadnicka A(2007)Volatile anesthetic-induced cardiac preconditioning J Anesth 21 212-219

[2] Marinovic J(2004)Mechanisms of cardioprotection by volatile anesthetics Anesthesiology 100 707-721

[3] Ljubkovic M(2004)Adenosine and a nitric oxide donor enhances cardioprotection by preconditioning with isoflurane through mitochondrial adenosine triphosphate-sensitive K+ channel-dependent and -independent mechanisms Anesthesiology 100 515-524

[4] Bienengraeber MW(2008)Sevoflurane enhances ethanol-induced cardiac preconditioning through modulation of protein kinase C, mitochondrial KATP channels, and nitric oxide synthase, in guinea pig hearts Anesth Analg 106 9-16

[5] Bosnjak ZJ(2009)Acute memory phase of sevoflurane preconditioning is associated with sustained translocation of protein kinase C-α and ε, but not δ, in isolated guinea pig hearts Eur J Anaesthesiol 26 582-43

[6] Tanaka K(2009)Aprotinin abolishes sevoflurane postconditioning by inhibiting nitric oxide production and phosphorylation of protein kinase C-δ and glycogen synthase kinase 3β Anesthesiology 111 1036-765

[7] Ludwig LM(2008)Postconditioning by volatile anesthetics: salvaging ischemic myocardium at reperfusion by activation of prosurvival signaling J Cardiothorac Vasc Anesth 22 753-90

[8] Kersten JR(2001)Progressively developed myocardial apoptotic cell death during late phase of reperfusion Apoptosis 6 279-79

[9] Pagel PS(2006)Immunohistochemical expression of activated caspase-3 in human myocardial infarction Virchows Arch 448 75-460

[10] Warltier DC(2004)New directions for protecting the heart against ischaemia-reperfusion injury: targeting the reperfusion injury salvage kinase (RISK)-pathway Cardiovasc Res 61 448-232