Reduced food intake in response to CGP 71683A may be due to mechanisms other than NPY Y5 receptor blockade

被引:39
作者
O Della Zuana
M Sadlo
M Germain
M Félétou
S Chamorro
F Tisserand
C de Montrion
JF Boivin
J Duhault
JA Boutin
N Levens
机构
[1] Metabolic Diseases,
[2] Institut de Recherches Servier,undefined
[3] Molecular and Cell Biology,undefined
[4] Institut de Recherches Servier,undefined
[5] Clinical Biology,undefined
[6] Institut de Recherches Servier,undefined
关键词
CGP 71683A; neuropeptide Y; food intake; obesity; serotonin receptors; muscarinic receptors; adrenergic receptors;
D O I
10.1038/sj.ijo.0801472
中图分类号
学科分类号
摘要
INTRODUCTION: The purpose of this study was to test the continuing validity of the hypothesis that neuropeptide Y (NPY) produced in the brain controls food intake through an interaction with the NPY Y5 receptor subtype.
引用
收藏
页码:84 / 94
页数:10
相关论文
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