Hypoxic Excitability Changes and Sodium Currents in Hippocampus CA1 Neurons

[1] Arabadzisz D.(2002)Increased inter-spike intervals and fast afterhyperpolarization of action potentials in rat hippocampal pyramidal cells accompanied with altered calbindin immunoreactivity 10-12 months after global forebrain ischemia Neurosci. Lett. 331 103-106

[2] Ylinen A.(1962)The effects of changes in internal ionic concentrations on the electrical properties of perfused giant axons J. Physiol. (Lond.) 164 355-374

[3] Emri Z.(1995)Internal Ca2+ stores involved in anoxic responses of rat hippocampal neurons J. Physiol. 486 547-556

[4] Baker P. F.(1993)Human neocortical excitability is decreased during anoxia via sodium channel modulation J. Clin. Invest. 91 608-615

[5] Hodgkin A. L.(1995)Hyperexcitability in CA1 of the rat hippocampal slice following hypoxia or adenosine Brain Res. 677 127-137

[6] Shaw T. I.(2001)Chemical hypoxia in hippocampal pyramidal cells affects membrane potential differentially depending on resting potentia Neuroscience 106 89-94

[7] Belousov A. B.(1998)Potassium conductance causing hyperpolarization of CA1 hippocampal neurons during hypoxia J. Neurophysiol. 80 2378-2390

[8] Godfraind J.-M.(2001)Enhanced spontaneous transmitter release is the earliest consequence of neocortical hypoxia that can explain the disruption of normal circuit function J. Neurosci. 21 4600-4608

[9] Krnjevic K.(1997)Contribution of ATPsensitive potassium channels to hypoxic hyperpolarization in rat hippocampal CA1 neurons in vitro J. Neurophysiol. 77 378-385

[10] Cummins T. R.(1987)Effects of hypoxia on rat hippocampal neurones in vitro J. Physiol. 384 131-151

[1] Arabadzisz D.(2002)Increased inter-spike intervals and fast afterhyperpolarization of action potentials in rat hippocampal pyramidal cells accompanied with altered calbindin immunoreactivity 10-12 months after global forebrain ischemia Neurosci. Lett. 331 103-106

[2] Ylinen A.(1962)The effects of changes in internal ionic concentrations on the electrical properties of perfused giant axons J. Physiol. (Lond.) 164 355-374

[3] Emri Z.(1995)Internal Ca2+ stores involved in anoxic responses of rat hippocampal neurons J. Physiol. 486 547-556

[4] Baker P. F.(1993)Human neocortical excitability is decreased during anoxia via sodium channel modulation J. Clin. Invest. 91 608-615

[5] Hodgkin A. L.(1995)Hyperexcitability in CA1 of the rat hippocampal slice following hypoxia or adenosine Brain Res. 677 127-137

[6] Shaw T. I.(2001)Chemical hypoxia in hippocampal pyramidal cells affects membrane potential differentially depending on resting potentia Neuroscience 106 89-94

[7] Belousov A. B.(1998)Potassium conductance causing hyperpolarization of CA1 hippocampal neurons during hypoxia J. Neurophysiol. 80 2378-2390

[8] Godfraind J.-M.(2001)Enhanced spontaneous transmitter release is the earliest consequence of neocortical hypoxia that can explain the disruption of normal circuit function J. Neurosci. 21 4600-4608

[9] Krnjevic K.(1997)Contribution of ATPsensitive potassium channels to hypoxic hyperpolarization in rat hippocampal CA1 neurons in vitro J. Neurophysiol. 77 378-385

[10] Cummins T. R.(1987)Effects of hypoxia on rat hippocampal neurones in vitro J. Physiol. 384 131-151