Direct demonstration in dogs of the absence of lethal reperfusion injury

Reimer K.A., Murry C.E., Richard V.J., The role of neutrophils and free radicals in the ischemic-reperfused heart

why the confusion and controversy?, J Mol Cell Cardiol, 21, pp. 1225-1239, (1989)

Hoffman M., Genth K., Schaper W., The influence of reperfusion on infarct size after experimental coronary artery occlusion, Basic Res Cardiol, 75, pp. 572-582, (1980)

Ganz W., Watanabe I., Kanamasa K., Yano J., Han D.S., Fishbein M.C., Does reperfusion extend necrosis? A study in a single territory of myocardial ischemia, half reperfused and half not reperfused, Circulation, 82, pp. 1020-1033, (1989)

Zahger D., Yano J., Chaux A., Fishbein M.C., Ganz W., Absence of lethal reperfusion injury after 3 hours of reperfusion, Circulation, 91, pp. 2989-2994, (1995)

Narula J., Nicol P.D., Southern J.F., Et al., Evaluation of myocardial infarct size before and after reperfusion: Dual tracer imaging with radiolabeled antimyosin antibody, J Nuclear Med, 35, pp. 1076-1085, (1994)

Farb A., Kolodgie F.D., Jenkins M., Virmani R., Myocardial infarct extension during reperfusion after coronary artery occlusion: Pathologic evidence, J Am Coll Cardiol, 21, pp. 1245-1253, (1993)

Khaw B.A., Strauss H.W., Moore R., Fallon J.T., Yasuda T., Gold H.K., Haber E., Myocardial damage delineated by 111-In antimyosin Fab and technetium-99m pyrophosphate, J Nuclear Med, 28, pp. 76-82, (1987)

Takeda K., Lafrance N.D., Weisman H.F., Wagner H.N., Becker L.C., Comparison of indium-111 antimyosin antibody and technetium-99m pyrophosphate localization in reperfused and nonreperfused myocardial infarction, J Am Coll Cardiol, 17, pp. 519-526, (1991)

Kent S.P., Intracellular plasma protein: A manifestation of cell injury in myocardial ischemia, Nature, 210, pp. 1279-1281, (1966)

Reimer K.A., Murry C.E., Richard V.J., The role of neutrophils and free radicals in the ischemic-reperfused heart

why the confusion and controversy?, J Mol Cell Cardiol, 21, pp. 1225-1239, (1989)

Hoffman M., Genth K., Schaper W., The influence of reperfusion on infarct size after experimental coronary artery occlusion, Basic Res Cardiol, 75, pp. 572-582, (1980)

Ganz W., Watanabe I., Kanamasa K., Yano J., Han D.S., Fishbein M.C., Does reperfusion extend necrosis? A study in a single territory of myocardial ischemia, half reperfused and half not reperfused, Circulation, 82, pp. 1020-1033, (1989)

Zahger D., Yano J., Chaux A., Fishbein M.C., Ganz W., Absence of lethal reperfusion injury after 3 hours of reperfusion, Circulation, 91, pp. 2989-2994, (1995)

Narula J., Nicol P.D., Southern J.F., Et al., Evaluation of myocardial infarct size before and after reperfusion: Dual tracer imaging with radiolabeled antimyosin antibody, J Nuclear Med, 35, pp. 1076-1085, (1994)

Farb A., Kolodgie F.D., Jenkins M., Virmani R., Myocardial infarct extension during reperfusion after coronary artery occlusion: Pathologic evidence, J Am Coll Cardiol, 21, pp. 1245-1253, (1993)

Khaw B.A., Strauss H.W., Moore R., Fallon J.T., Yasuda T., Gold H.K., Haber E., Myocardial damage delineated by 111-In antimyosin Fab and technetium-99m pyrophosphate, J Nuclear Med, 28, pp. 76-82, (1987)

Takeda K., Lafrance N.D., Weisman H.F., Wagner H.N., Becker L.C., Comparison of indium-111 antimyosin antibody and technetium-99m pyrophosphate localization in reperfused and nonreperfused myocardial infarction, J Am Coll Cardiol, 17, pp. 519-526, (1991)

Kent S.P., Intracellular plasma protein: A manifestation of cell injury in myocardial ischemia, Nature, 210, pp. 1279-1281, (1966)