TRANSIENT ISCHEMIA LEADS TO INTRACELLULAR ALKALOSIS IN THE BRAIN

被引：9

作者：

BLOMQVIST, P

MABE, H

SIESJO, BK

机构：

来源：

ACTA PHYSIOLOGICA SCANDINAVICA | 1982年 / 116卷 / 01期

关键词：

D O I：

10.1111/j.1748-1716.1982.tb10606.x

中图分类号：

Q4 [生理学];

学科分类号：

071003 ;

摘要：

引用

收藏

页码：103 / 104

页数：2

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[1] INFLUENCE OF TISSUE ACIDOSIS UPON RESTITUTION OF BRAIN ENERGY METABOLISM FOLLOWING TOTAL ISCHEMIA
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[2] CHANGES IN ENERGY STATE AND ACID-BASE PARAMETERS OF RAT-BRAIN DURING COMPLETE COMPRESSION ISCHEMIA
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[J]. BRAIN RESEARCH, 1974, 73 (02) : 277 - 289
[3] EFFECTS OF PHENOBARBITAL IN CEREBRAL ISCHEMIA .2. RESTITUTION OF CEREBRAL ENERGY-STATE, AS WELL AS OF GLYCOLYTIC METABOLITES, CITRIC-ACID CYCLE INTERMEDIATES AND ASSOCIATED AMINO-ACIDS AFTER PRONOUNCED INCOMPLETE ISCHEMIA
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[J]. STROKE, 1978, 9 (04) : 335 - 343
[4] NEW MODEL OF BILATERAL HEMISPHERIC ISCHEMIA IN THE UNANESTHETIZED RAT
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[J]. STROKE, 1979, 10 (03) : 267 - 272
[5] EFFECT OF HYPERCAPNIA UPON INTRACELLULAR PH IN BRAIN, EVALUATED BY BICARBONATE CARBONIC ACID METHOD AND FROM CREATINE-PHOSPHOKINASE EQUILIBRIUM
SIESJO, BK
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