OXIDATION OF UROPORPHYRINOGENS BY HYDROXYL RADICALS - EVIDENCE FOR NONPORPHYRIN PRODUCTS AS POTENTIAL INHIBITORS OF UROPORPHYRINOGEN DECARBOXYLASE

被引：54

作者：

FRANCIS, JE ^{[1
]}

SMITH, AG ^{[1
]}

机构：

[1] MRC LABS,TOXICOL UNIT,WOODMANSTERNE RD,CARSHALTON SM5 4EF,SURREY,ENGLAND

来源：

FEBS LETTERS | 1988年 / 233卷 / 02期

关键词：

D O I：

10.1016/0014-5793(88)80449-6

中图分类号：

Q5 [生物化学]; Q7 [分子生物学];

学科分类号：

071010 ; 081704 ;

摘要：

引用

页码：311 / 314

页数：4

共 20 条

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[4] INCREASED OXIDATION OF UROPORPHYRINOGEN BY AN INDUCIBLE LIVER MICROSOMAL SYSTEM - POSSIBLE RELEVANCE TO DRUG-INDUCED UROPORPHYRIA
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[5] DRUG-INDUCED ACCUMULATION OF UROPORPHYRIN IN CHICKEN HEPATOCYTE CULTURES - STRUCTURAL REQUIREMENTS FOR THE EFFECT AND ROLE OF EXOGENOUS IRON
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[6] ASSAY OF MOUSE-LIVER UROPORPHYRINOGEN DECARBOXYLASE BY REVERSE-PHASE HIGH-PERFORMANCE LIQUID-CHROMATOGRAPHY
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[J]. ANALYTICAL BIOCHEMISTRY, 1984, 138 (02) : 404 - 410
[7] POLYCYCLIC AROMATIC-HYDROCARBONS CAUSE HEPATIC PORPHYRIA IN IRON-LOADED C57BL-10 MICE - COMPARISON OF UROPORPHYRINOGEN DECARBOXYLASE INHIBITION WITH INDUCTION OF ALKOXYPHENOXAZONE DEALKYLATIONS
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[J]. BIOCHEMICAL AND BIOPHYSICAL RESEARCH COMMUNICATIONS, 1987, 146 (01) : 13 - 20
[8] GRANICK S, 1975, J BIOL CHEM, V250, P9215
[9] OXYGEN-TOXICITY, OXYGEN RADICALS, TRANSITION-METALS AND DISEASE
HALLIWELL, B
GUTTERIDGE, JMC
[J]. BIOCHEMICAL JOURNAL, 1984, 219 (01) : 1 - 14
[10] EXPOSURE TO TOXIC AGENTS - THE HEME BIOSYNTHETIC-PATHWAY AND HEMOPROTEINS AS INDICATOR
MARKS, GS
[J]. CRC CRITICAL REVIEWS IN TOXICOLOGY, 1985, 15 (02): : 151 - 179

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