P53 IN THE DIAGNOSIS OF HUMAN NEOPLASIA

被引：35

作者：

COSSMAN, J

SCHLEGEL, R

机构：

来源：

JOURNAL OF THE NATIONAL CANCER INSTITUTE | 1991年 / 83卷 / 14期

关键词：

D O I：

10.1093/jnci/83.14.980

中图分类号：

R73 [肿瘤学];

学科分类号：

100214 ;

摘要：

引用

页码：980 / 981

页数：2

共 21 条

[1] THE MOLECULAR-GENETICS OF CANCER [J].

BISHOP, JM .

SCIENCE, 1987, 235 (4786) :305-311

[2] PARTICIPATION OF P53 CELLULAR TUMOR-ANTIGEN IN TRANSFORMATION OF NORMAL EMBRYONIC-CELLS [J].

ELIYAHU, D ;

RAZ, A ;

GRUSS, P ;

GIVOL, D ;

OREN, M .

NATURE, 1984, 312 (5995) :646-649

[3] OVERPRODUCTION OF P53 ANTIGEN MAKES ESTABLISHED CELLS HIGHLY TUMORIGENIC [J].

ELIYAHU, D ;

MICHALOVITZ, D ;

OREN, M .

NATURE, 1985, 316 (6024) :158-160

[4] WILD-TYPE P53 CAN INHIBIT ONCOGENE-MEDIATED FOCUS FORMATION [J].

ELIYAHU, D ;

MICHALOVITZ, D ;

ELIYAHU, S ;

PINHASIKIMHI, O ;

OREN, M .

PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA, 1989, 86 (22) :8763-8767

[5]

ELIYAHU D, 1988, ONCOGENE, V3, P313

[6] A GENETIC MODEL FOR COLORECTAL TUMORIGENESIS [J].

FEARON, ER ;

VOGELSTEIN, B .

CELL, 1990, 61 (05) :759-767

[7] THE P53 PROTO-ONCOGENE CAN ACT AS A SUPPRESSOR OF TRANSFORMATION [J].

FINLAY, CA ;

HINDS, PW ;

LEVINE, AJ .

CELL, 1989, 57 (07) :1083-1093

[8]

FRIEND S, 1990, MOL GENETICS DIAGNOS, P20

[9] MUTATION IS REQUIRED TO ACTIVATE THE P53 GENE FOR COOPERATION WITH THE RAS ONCOGENE AND TRANSFORMATION [J].

HINDS, P ;

FINLAY, C ;

LEVINE, AJ .

JOURNAL OF VIROLOGY, 1989, 63 (02) :739-746

[10] CELLULAR IMMORTALIZATION BY A CDNA CLONE ENCODING THE TRANSFORMATION-ASSOCIATED PHOSPHOPROTEIN P53 [J].

JENKINS, JR ;

RUDGE, K ;

CURRIE, GA .

NATURE, 1984, 312 (5995) :651-654

← 1 2 3 →