ATHEROSCLEROSIS - SCAVENGER CELL-RECEPTOR SHARED

[1] BLOMHOFF R, 1984, J BIOL CHEM, V259, P8898

[2] LIPOPROTEIN METABOLISM IN THE MACROPHAGE - IMPLICATIONS FOR CHOLESTEROL DEPOSITION IN ATHEROSCLEROSIS [J]. ANNUAL REVIEW OF BIOCHEMISTRY, 1983, 52 : 223 - 261

[3] MALONDIALDEHYDE ALTERATION OF LOW-DENSITY LIPOPROTEINS LEADS TO CHOLESTERYL ESTER ACCUMULATION IN HUMAN MONOCYTE-MACROPHAGES [J]. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA-BIOLOGICAL SCIENCES, 1980, 77 (04): : 2214 - 2218

[4] BINDING-SITE ON MACROPHAGES THAT MEDIATES UPTAKE AND DEGRADATION OF ACETYLATED LOW-DENSITY LIPOPROTEIN, PRODUCING MASSIVE CHOLESTEROL DEPOSITION [J]. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA, 1979, 76 (01) : 333 - 337

[5] HIGH-AFFINITY UPTAKE AND DEGRADATION OF ACETYLATED LOW-DENSITY LIPOPROTEIN BY CONFLUENT HUMAN VASCULAR ENDOTHELIAL-CELLS [J]. ATHEROSCLEROSIS, 1985, 56 (01) : 81 - 92

[6] HUANG SS, 1982, J BIOL CHEM, V257, P1546

[7] NAGELKERKE JF, 1983, J BIOL CHEM, V258, P2221

[8] ESSENTIAL ROLE OF PHOSPHOLIPASE-A2 ACTIVITY IN ENDOTHELIAL CELL-INDUCED MODIFICATION OF LOW-DENSITY LIPOPROTEIN [J]. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA, 1985, 82 (09) : 3000 - 3004

[9] PLATELET SECRETORY PRODUCTS INHIBIT LIPOPROTEIN METABOLISM IN MACROPHAGES [J]. NATURE, 1985, 316 (6030) : 746 - 748

[10] UPTAKE OF CHEMICALLY MODIFIED LOW-DENSITY LIPOPROTEINS INVIVO IS MEDIATED BY SPECIFIC ENDOTHELIAL-CELLS [J]. JOURNAL OF CELL BIOLOGY, 1985, 100 (01) : 103 - 117

[1] BLOMHOFF R, 1984, J BIOL CHEM, V259, P8898

[2] LIPOPROTEIN METABOLISM IN THE MACROPHAGE - IMPLICATIONS FOR CHOLESTEROL DEPOSITION IN ATHEROSCLEROSIS [J]. ANNUAL REVIEW OF BIOCHEMISTRY, 1983, 52 : 223 - 261

[3] MALONDIALDEHYDE ALTERATION OF LOW-DENSITY LIPOPROTEINS LEADS TO CHOLESTERYL ESTER ACCUMULATION IN HUMAN MONOCYTE-MACROPHAGES [J]. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA-BIOLOGICAL SCIENCES, 1980, 77 (04): : 2214 - 2218

[4] BINDING-SITE ON MACROPHAGES THAT MEDIATES UPTAKE AND DEGRADATION OF ACETYLATED LOW-DENSITY LIPOPROTEIN, PRODUCING MASSIVE CHOLESTEROL DEPOSITION [J]. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA, 1979, 76 (01) : 333 - 337

[5] HIGH-AFFINITY UPTAKE AND DEGRADATION OF ACETYLATED LOW-DENSITY LIPOPROTEIN BY CONFLUENT HUMAN VASCULAR ENDOTHELIAL-CELLS [J]. ATHEROSCLEROSIS, 1985, 56 (01) : 81 - 92

[6] HUANG SS, 1982, J BIOL CHEM, V257, P1546

[7] NAGELKERKE JF, 1983, J BIOL CHEM, V258, P2221

[8] ESSENTIAL ROLE OF PHOSPHOLIPASE-A2 ACTIVITY IN ENDOTHELIAL CELL-INDUCED MODIFICATION OF LOW-DENSITY LIPOPROTEIN [J]. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA, 1985, 82 (09) : 3000 - 3004

[9] PLATELET SECRETORY PRODUCTS INHIBIT LIPOPROTEIN METABOLISM IN MACROPHAGES [J]. NATURE, 1985, 316 (6030) : 746 - 748

[10] UPTAKE OF CHEMICALLY MODIFIED LOW-DENSITY LIPOPROTEINS INVIVO IS MEDIATED BY SPECIFIC ENDOTHELIAL-CELLS [J]. JOURNAL OF CELL BIOLOGY, 1985, 100 (01) : 103 - 117