COMPLEMENT AND HOST DEFENSE AGAINST MICROORGANISMS

[1] AGNELLO V, 1978, MEDICINE, V57, P1

[2] ALPER CA, 1972, LANCET, V2, P1179

[3] STUDIES IN-VIVO AND IN-VITRO ON AN ABNORMALITY IN METABOLISM OF C3 IN A PATIENT WITH INCREASED SUSCEPTIBILITY TO INFECTION [J]. JOURNAL OF CLINICAL INVESTIGATION, 1970, 49 (11) : 1975 - &

[4] HOMOZYGOUS HUMAN C3 DEFICIENCY - ROLE OF C3 IN ANTIBODY-PRODUCTION, C1S-INDUCED VASOPERMEABILITY, AND COBRA VENOM-INDUCED PASSIVE HEMOLYSIS [J]. JOURNAL OF CLINICAL INVESTIGATION, 1976, 57 (01) : 222 - 229

[5] INCREASED SUSCEPTIBILITY TO INFECTION ASSOCIATED WITH ABNORMALITIES OF COMPLEMENT-MEDIATED FUNCTIONS AND OF THIRD COMPONENT OF COMPLEMENT (C3) [J]. NEW ENGLAND JOURNAL OF MEDICINE, 1970, 282 (07) : 349 - &

[6] ABNORMALITIES OF POLYMORPHONUCLEAR LEUKOCYTE FUNCTION ASSOCIATED WITH A HERITABLE DEFICIENCY OF HIGH MOLECULAR-WEIGHT SURFACE GLYCOPROTEINS (GP138) - COMMON RELATIONSHIP TO DIMINISHED CELL ADHERENCE [J]. JOURNAL OF CLINICAL INVESTIGATION, 1984, 74 (02) : 536 - 551

[7] INHIBITION OF PHAGOCYTOSIS OF COMPLEMENT-C3-COATED OR IMMUNOGLOBULIN-G-COATED PARTICLES AND OF C3BI BINDING BY MONOCLONAL-ANTIBODIES TO A MONOCYTE-GRANULOCYTE MEMBRANE GLYCOPROTEIN (MOL) [J]. JOURNAL OF CLINICAL INVESTIGATION, 1983, 72 (01) : 171 - 179

[8] ARNAOUT MA, 1981, J IMMUNOL, V127, P1348

[9] DEFICIENCY OF A GRANULOCYTE-MEMBRANE GLYCOPROTEIN (GP150) IN A BOY WITH RECURRENT BACTERIAL-INFECTIONS [J]. NEW ENGLAND JOURNAL OF MEDICINE, 1982, 306 (12) : 693 - 699

[10] ANTIBODY-INDEPENDENT CLASSICAL PATHWAY-MEDIATED OPSONOPHAGOCYTOSIS OF TYPE-IA, GROUP-B STREPTOCOCCUS [J]. JOURNAL OF CLINICAL INVESTIGATION, 1982, 69 (02) : 394 - 404

[1] AGNELLO V, 1978, MEDICINE, V57, P1

[2] ALPER CA, 1972, LANCET, V2, P1179

[3] STUDIES IN-VIVO AND IN-VITRO ON AN ABNORMALITY IN METABOLISM OF C3 IN A PATIENT WITH INCREASED SUSCEPTIBILITY TO INFECTION [J]. JOURNAL OF CLINICAL INVESTIGATION, 1970, 49 (11) : 1975 - &

[4] HOMOZYGOUS HUMAN C3 DEFICIENCY - ROLE OF C3 IN ANTIBODY-PRODUCTION, C1S-INDUCED VASOPERMEABILITY, AND COBRA VENOM-INDUCED PASSIVE HEMOLYSIS [J]. JOURNAL OF CLINICAL INVESTIGATION, 1976, 57 (01) : 222 - 229

[5] INCREASED SUSCEPTIBILITY TO INFECTION ASSOCIATED WITH ABNORMALITIES OF COMPLEMENT-MEDIATED FUNCTIONS AND OF THIRD COMPONENT OF COMPLEMENT (C3) [J]. NEW ENGLAND JOURNAL OF MEDICINE, 1970, 282 (07) : 349 - &

[6] ABNORMALITIES OF POLYMORPHONUCLEAR LEUKOCYTE FUNCTION ASSOCIATED WITH A HERITABLE DEFICIENCY OF HIGH MOLECULAR-WEIGHT SURFACE GLYCOPROTEINS (GP138) - COMMON RELATIONSHIP TO DIMINISHED CELL ADHERENCE [J]. JOURNAL OF CLINICAL INVESTIGATION, 1984, 74 (02) : 536 - 551

[7] INHIBITION OF PHAGOCYTOSIS OF COMPLEMENT-C3-COATED OR IMMUNOGLOBULIN-G-COATED PARTICLES AND OF C3BI BINDING BY MONOCLONAL-ANTIBODIES TO A MONOCYTE-GRANULOCYTE MEMBRANE GLYCOPROTEIN (MOL) [J]. JOURNAL OF CLINICAL INVESTIGATION, 1983, 72 (01) : 171 - 179

[8] ARNAOUT MA, 1981, J IMMUNOL, V127, P1348

[9] DEFICIENCY OF A GRANULOCYTE-MEMBRANE GLYCOPROTEIN (GP150) IN A BOY WITH RECURRENT BACTERIAL-INFECTIONS [J]. NEW ENGLAND JOURNAL OF MEDICINE, 1982, 306 (12) : 693 - 699

[10] ANTIBODY-INDEPENDENT CLASSICAL PATHWAY-MEDIATED OPSONOPHAGOCYTOSIS OF TYPE-IA, GROUP-B STREPTOCOCCUS [J]. JOURNAL OF CLINICAL INVESTIGATION, 1982, 69 (02) : 394 - 404