GLUCOSE CYCLING IN ISLETS FROM HEALTHY AND DIABETIC RATS

[1] LIVER GLUCOSE-6-PHOSPHATASE AND PYROPHOSPHATE-GLUCOSE PHOSPHOTRANSFERASE - EFFECTS OF FASTING [J]. BIOCHEMICAL AND BIOPHYSICAL RESEARCH COMMUNICATIONS, 1965, 20 (05) : 606 - &

[2] ASHCROFT FM, 1988, ANNU REV NEUROSCI, V11, P97, DOI 10.1146/annurev.ne.11.030188.000525

[3] GLUCORECEPTOR MECHANISMS AND THE CONTROL OF INSULIN RELEASE AND BIOSYNTHESIS [J]. DIABETOLOGIA, 1980, 18 (01) : 5 - 15

[4] RESPONSES OF NEONATAL RAT ISLETS TO STREPTOZOTOCIN - LIMITED B-CELL REGENERATION AND HYPERGLYCEMIA [J]. DIABETES, 1981, 30 (01) : 64 - 69

[5] COLCA JR, 1988, DIABETES S1, V37, pA7

[6] GLUCOSE-UTILIZATION IN ISLETS OF HYPERGLYCEMIC RAT MODELS WITH IMPAIRED GLUCOSE-INDUCED INSULIN-SECRETION [J]. METABOLISM-CLINICAL AND EXPERIMENTAL, 1987, 36 (04): : 335 - 337

[7] MILD TYPE-2 DIABETES MARKEDLY INCREASES GLUCOSE CYCLING IN THE POSTABSORPTIVE STATE AND DURING GLUCOSE-INFUSION IRRESPECTIVE OF OBESITY [J]. JOURNAL OF CLINICAL INVESTIGATION, 1988, 81 (06) : 1953 - 1961

[8] ASPECTS OF THE PATHOGENESIS OF TYPE-2 DIABETES [J]. ENDOCRINE REVIEWS, 1984, 5 (03) : 395 - 410

[9] HEXOSE METABOLISM IN PANCREATIC-ISLETS - ABSENCE OF GLUCOSE-6-PHOSPHATASE IN RAT ISLET CELLS [J]. MOLECULAR AND CELLULAR ENDOCRINOLOGY, 1987, 49 (2-3) : 219 - 225

[10] ELEVATED PROINSULIN BIOSYNTHESIS INVITRO FROM A RAT MODEL OF NON-INSULIN-DEPENDENT DIABETES-MELLITUS [J]. DIABETES, 1983, 32 (03) : 277 - 283

[1] LIVER GLUCOSE-6-PHOSPHATASE AND PYROPHOSPHATE-GLUCOSE PHOSPHOTRANSFERASE - EFFECTS OF FASTING [J]. BIOCHEMICAL AND BIOPHYSICAL RESEARCH COMMUNICATIONS, 1965, 20 (05) : 606 - &

[2] ASHCROFT FM, 1988, ANNU REV NEUROSCI, V11, P97, DOI 10.1146/annurev.ne.11.030188.000525

[3] GLUCORECEPTOR MECHANISMS AND THE CONTROL OF INSULIN RELEASE AND BIOSYNTHESIS [J]. DIABETOLOGIA, 1980, 18 (01) : 5 - 15

[4] RESPONSES OF NEONATAL RAT ISLETS TO STREPTOZOTOCIN - LIMITED B-CELL REGENERATION AND HYPERGLYCEMIA [J]. DIABETES, 1981, 30 (01) : 64 - 69

[5] COLCA JR, 1988, DIABETES S1, V37, pA7

[6] GLUCOSE-UTILIZATION IN ISLETS OF HYPERGLYCEMIC RAT MODELS WITH IMPAIRED GLUCOSE-INDUCED INSULIN-SECRETION [J]. METABOLISM-CLINICAL AND EXPERIMENTAL, 1987, 36 (04): : 335 - 337

[7] MILD TYPE-2 DIABETES MARKEDLY INCREASES GLUCOSE CYCLING IN THE POSTABSORPTIVE STATE AND DURING GLUCOSE-INFUSION IRRESPECTIVE OF OBESITY [J]. JOURNAL OF CLINICAL INVESTIGATION, 1988, 81 (06) : 1953 - 1961

[8] ASPECTS OF THE PATHOGENESIS OF TYPE-2 DIABETES [J]. ENDOCRINE REVIEWS, 1984, 5 (03) : 395 - 410

[9] HEXOSE METABOLISM IN PANCREATIC-ISLETS - ABSENCE OF GLUCOSE-6-PHOSPHATASE IN RAT ISLET CELLS [J]. MOLECULAR AND CELLULAR ENDOCRINOLOGY, 1987, 49 (2-3) : 219 - 225

[10] ELEVATED PROINSULIN BIOSYNTHESIS INVITRO FROM A RAT MODEL OF NON-INSULIN-DEPENDENT DIABETES-MELLITUS [J]. DIABETES, 1983, 32 (03) : 277 - 283