TUBULAR NA, K-ATPASE DEFICIENCY, CAUSE OF CONGENITAL RENAL SALT-LOSING SYNDROME

被引:35
作者
BIERICH, JR
SCHMIDT, U
机构
[1] UNIV TUBINGEN,KINDER KLIN,RUMELIN STR 23,D-7400 TUBINGEN,FED REP GER
[2] UNIV TUBINGEN,PATHOL INST,RUMELIN STR 23,D-7400 TUBINGEN,FED REP GER
关键词
D O I
10.1007/BF00443063
中图分类号
R72 [儿科学];
学科分类号
100202 ;
摘要
引用
收藏
页码:81 / 87
页数:7
相关论文
共 6 条
[1]  
MULLER M, IN PRESS
[2]   FUNCTION OF NA, K-ATPASE IN SINGLE PORTIONS OF RAT NEPHRON [J].
SCHMIDT, U ;
SCHMID, H ;
FUNK, B ;
DUBACH, UC .
ANNALS OF THE NEW YORK ACADEMY OF SCIENCES, 1974, 242 (NOV29) :489-500
[3]   ACTIVITY OF (NA+K+)-STIMULATED ADENOSINTRIPHOSPHATASE IN RAT NEPHRON [J].
SCHMIDT, U ;
DUBACH, UC .
PFLUGERS ARCHIV-EUROPEAN JOURNAL OF PHYSIOLOGY, 1969, 306 (03) :219-&
[4]  
THURAU K, 1966, HDB KINDERHEILKUNDE, V7, P993
[5]   ALDOSTERONE BIOSYNTHETIC DEFECT IN SALT-LOSING DISORDER [J].
ULICK, S ;
YAFFE, S ;
GAUTIER, E ;
MARKELLO, JR ;
LOWE, CU ;
VETTER, KK .
JOURNAL OF CLINICAL ENDOCRINOLOGY & METABOLISM, 1964, 24 (07) :669-+
[6]   NEW HEREDITARY DEFECT IN BIOSYNTHESIS OF ALDOSTERONE - URINARY C21-CORTICOSTEROID PATTERN IN 3 RELATED PATIENTS WITH SALT-LOSING SYNDROME SUGGESTING 18-OXIDATION DEFECT [J].
VISSER, HKA ;
COST, WS .
ACTA ENDOCRINOLOGICA, 1964, 47 (04) :589-&