LETHAL TOXICITY OF LIPOPOLYSACCHARIDE AND TUMOR-NECROSIS-FACTOR IN NORMAL AND D-GALACTOSAMINE-TREATED MICE

被引：547

作者：

LEHMANN, V

FREUDENBERG, MA

GALANOS, C

机构：

[1] MAX PLANCK INST IMMUNBIOL,STUBEWEG 51,D-7800 FREIBURG,FED REP GER

[2] DEUTSCH KREBSFORSCHUNGSZENTRUM,D-6900 HEIDELBERG,FED REP GER

来源：

JOURNAL OF EXPERIMENTAL MEDICINE | 1987年 / 165卷 / 03期

关键词：

D O I：

10.1084/jem.165.3.657

中图分类号：

R392 [医学免疫学]; Q939.91 [免疫学];

学科分类号：

100102 ;

摘要：

引用

页码：657 / 663

页数：7

共 16 条

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[2] PASSIVE-IMMUNIZATION AGAINST CACHECTIN TUMOR NECROSIS FACTOR PROTECTS MICE FROM LETHAL EFFECT OF ENDOTOXIN
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[5] GALACTOSAMINE HEPATITIS - KEY ROLE OF THE NUCLEOTIDE DEFICIENCY PERIOD IN THE PATHOGENESIS OF CELL INJURY AND CELL-DEATH
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[6] TUMOR-NECROSIS-FACTOR (CACHECTIN) IS AN ENDOGENOUS PYROGEN AND INDUCES PRODUCTION OF INTERLEUKIN-1
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[7] REQUIREMENT FOR LIPOPOLYSACCHARIDE-RESPONSIVE MACROPHAGES IN GALACTOSAMINE-INDUCED SENSITIZATION TO ENDOTOXIN
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[J]. INFECTION AND IMMUNITY, 1986, 51 (03) : 891 - 895
[8] GALANOS C, 1979, ZBL BAKT-INT J MED M, V243, P226
[9] GALACTOSAMINE-INDUCED SENSITIZATION TO THE LETHAL EFFECTS OF ENDOTOXIN
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[J]. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA, 1979, 76 (11) : 5939 - 5943
[10] LIPOPROTEIN-LIPASE SUPPRESSION IN 3T3-L1 CELLS BY AN ENDOTOXIN-INDUCED MEDIATOR FROM EXUDATE CELLS
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[J]. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA-BIOLOGICAL SCIENCES, 1982, 79 (03): : 912 - 916

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