INTERLEUKIN-1 DECREASES RENAL SODIUM-REABSORPTION - POSSIBLE MECHANISM OF ENDOTOXIN-INDUCED NATRIURESIS

被引：64

作者：

CAVERZASIO, J ^{[1
]}

RIZZOLI, R ^{[1
]}

DAYER, JM ^{[1
]}

BONJOUR, JP ^{[1
]}

机构：

[1] UNIV GENEVA,DEPT MED,DIV IMMUNOL & ALLERGY,CH-1211 GENEVA 4,SWITZERLAND

来源：

AMERICAN JOURNAL OF PHYSIOLOGY | 1987年 / 252卷 / 05期

关键词：

D O I：

10.1152/ajprenal.1987.252.5.F943

中图分类号：

Q4 [生理学];

学科分类号：

071003 ;

摘要：

引用

页码：F943 / F946

页数：4

共 12 条

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[3] DINARELLO CA, 1984, NEW ENGL J MED, V311, P1413
[4] GAGALO IT, 1986, PHARMACOL RES, V18, P17
[5] GANGULI M, 1977, CIRC RES, V40, P135
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[7] RENAL-FUNCTION IN CONSCIOUS RATS AFTER INDOMETHACIN - EVIDENCE FOR A TUBULAR ACTION OF ENDOGENOUS PROSTAGLANDINS
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[8] RENAL EXCRETION OF PROSTAGLANDIN METABOLITES, ARGININE VASOPRESSIN, AND SODIUM DURING ENDOTOXIN AND ENDOGENOUS PYROGEN INDUCED FEVER IN THE GOAT
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[9] THERE IS MORE THAN ONE INTERLEUKIN-1
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