EXPOSURE OF DOG THYROID SLICES TO ACETYLCHOLINE INDUCES REFRACTORINESS TO ITS SUBSEQUENT STIMULATION OF GLUCOSE-OXIDATION

被引：6

作者：

AREM, R

CHAYOTH, R

SHENKENBERG, TD

MILLER, SI

CHOU, MCY

FIELD, JB

机构：

[1] ST LUKES EPISCOPAL HOSP, DIABET RES LAB, HOUSTON, TX 77030 USA

[2] BAYLOR COLL MED, TEXAS MED CTR, DEPT MED, DIV ENDOCRINOL & METAB, HOUSTON, TX 77030 USA

来源：

ARCHIVES OF BIOCHEMISTRY AND BIOPHYSICS | 1983年 / 225卷 / 01期

关键词：

D O I：

10.1016/0003-9861(83)90007-3

中图分类号：

Q5 [生物化学]; Q7 [分子生物学];

学科分类号：

071010 ; 081704 ;

摘要：

引用

页码：66 / 74

页数：9

共 39 条

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[6] VOLTAGE CLAMP ANALYSIS OF ACETYLCHOLINE-RECEPTOR DESENSITIZATION IN ISOLATED MOLLUSK NEURONS
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[9] ROLE OF CALCIUM AND GUANOSINE 3'-5'-MONOPHOSPHATE IN THE ACTION OF ACETYLCHOLINE ON THYROID METABOLISM
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[J]. EUROPEAN JOURNAL OF BIOCHEMISTRY, 1980, 104 (01): : 199 - 208
[10] ACTIVATION, INACTIVATION, AND DESENSITIZATION OF ACETYLCHOLINE-RECEPTOR CHANNEL COMPLEX DETECTED BY BINDING OF PERHYDROHISTRIONICOTOXIN
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[J]. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA-BIOLOGICAL SCIENCES, 1980, 77 (04): : 2309 - 2313

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