ANTIDROMIC VASODILATATION AND NEUROGENIC INFLAMMATION

被引：4

作者：

CHAHL, LA ^{[1
]}

SZOLCSANYI, J ^{[1
]}

机构：

[1] UNIV PECS, SCH MED, INST PHARMACOL, H-7643 PECS, HUNGARY

来源：

NEUROSCIENCE LETTERS | 1983年 / 42卷 / 02期

关键词：

D O I：

10.1016/0304-3940(83)90410-X

中图分类号：

Q189 [神经科学];

学科分类号：

071006 ;

摘要：

引用

页码：219 / 225

页数：7

共 13 条

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[2] EVIDENCE THAT THE CONTRACTILE RESPONSE OF THE GUINEA-PIG ILEUM TO CAPSAICIN IS DUE TO SUBSTANCE-P RELEASE [J].

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[5] PHARMACOLOGICALLY INDUCED SELECTIVE DEGENERATION OF CHEMOSENSITIVE PRIMARY SENSORY NEURONS [J].

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KIRALY, E ;

JANCSOGABOR, A .

NATURE, 1977, 270 (5639) :741-743

[6] ROLE OF SENSORY NERVE ENDINGS IN NEUROGENIC INFLAMMATION INDUCED IN HUMAN SKIN AND IN EYE AND PAW OF RAT [J].

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[7] DIRECT EVIDENCE FOR NEUROGENIC INFLAMMATION AND ITS PREVENTION BY DENERVATION AND BY PRETREATMENT WITH CAPSAICIN [J].

JANCSO, N ;

JANCSOGA.A ;

SZOLCSANYI, J .

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[8] CAPSAICIN-INDUCED DEPLETION OF SUBSTANCE-P FROM PRIMARY SENSORY NEURONS [J].

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IVERSEN, LL ;

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[9] INHIBITION OF NEUROGENIC VASODILATATION AND PLASMA EXTRAVASATION BY SUBSTANCE-P ANTAGONISTS, SOMATOSTATIN AND [D-MET2,PRO5]ENKEPHALINAMIDE [J].

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[10]

Lewis T, 1927, HEART-J STUD CIRC, V14, P27

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