ASSOCIATION OF A COMPLEMENT ALLOTYPE (C3F) WITH ACUTE INFLAMMATORY RESPONSES TO CANDIDA-ALBICANS INFECTION

被引:4
作者
ASHMAN, RB [1 ]
KAY, PH [1 ]
LYNCH, DM [1 ]
OTT, K [1 ]
机构
[1] KING EDWARD MEM HOSP WOMEN,SUBIACO,WA 6008,AUSTRALIA
关键词
D O I
10.5694/j.1326-5377.1994.tb125927.x
中图分类号
R5 [内科学];
学科分类号
1002 ; 100201 ;
摘要
引用
收藏
页码:732 / 733
页数:2
相关论文
共 9 条
[1]   GENETIC POLYMORPHISM IN HUMAN GLYCINE-RICH BETA-GLYCOPROTEIN [J].
ALPER, CA ;
BOENISCH, T ;
WATSON, L .
JOURNAL OF EXPERIMENTAL MEDICINE, 1972, 135 (01) :68-&
[2]   CAPACITY OF COMPLEMENT C3 PHENOTYPES TO BIND ON TO MONONUCLEAR-CELLS IN MAN [J].
ARVILOMMI, H .
NATURE, 1974, 251 (5477) :740-741
[3]   COMPLEMENT-DERIVED PEPTIDE MEDIATORS OF INFLAMMATION [J].
JOSE, PJ .
BRITISH MEDICAL BULLETIN, 1987, 43 (02) :336-349
[4]   WHATS NEW IN THE ROLE OF COMPLEMENT IN DISEASES [J].
KAY, PH ;
PAPADIMITRIOU, JM .
PATHOLOGY RESEARCH AND PRACTICE, 1990, 186 (03) :410-414
[5]   DIFFERENT ALLOTYPES OF C3-DEGRADE AT DIFFERENT RATES [J].
KAY, PH ;
NATSUUMESAKAI, S ;
HAYAKAWA, J ;
DAWKINS, RL .
IMMUNOGENETICS, 1985, 22 (06) :563-569
[6]  
MEECH RJ, 1985, NEW ZEAL MED J, V98, P1
[7]  
ODDS FC, 1988, J MED VET MYCOL, V26, P277
[8]   GENES ASSOCIATED WITH RHEUMATOID-ARTHRITIS AND MILD INFLAMMATORY ARTHRITIS .2. ASSOCIATION OF HLA WITH COMPLEMENT C3 AND IMMUNOGLOBULIN GM ALLOTYPES [J].
PUTTICK, AH ;
BRIGGS, DC ;
WELSH, KI ;
WILLIAMSON, EA ;
JACOBY, RK ;
JONES, VE .
ANNALS OF THE RHEUMATIC DISEASES, 1990, 49 (04) :225-228
[9]   ACTIVATION OF ALTERNATIVE (PROPERDIN) PATHWAY OF COMPLEMENT BY CANDIDA-ALBICANS AND RELATED SPECIES [J].
RAY, TL ;
WUEPPER, KD .
JOURNAL OF INVESTIGATIVE DERMATOLOGY, 1976, 67 (06) :700-703