PROLYL HYDROXYLASE AND COLLAGEN-METABOLISM AFTER EXPERIMENTAL MYOCARDIAL-INFARCTION

被引：36

作者：

JUDD, JT

WEXLER, BC

机构：

[1] JEWISH HOSP,MAY INST MED RES,CINCINNATI,OH

[2] UNIV CINCINNATI,COLL MED,DEPT PATHOL,CINCINNATI,OH 45219

[3] UNIV CINCINNATI,COLL MED,DEPT MED,CINCINNATI,OH 45219

来源：

AMERICAN JOURNAL OF PHYSIOLOGY | 1975年 / 228卷 / 01期

关键词：

D O I：

10.1152/ajplegacy.1975.228.1.212

中图分类号：

Q4 [生理学];

学科分类号：

071003 ;

摘要：

引用

页码：212 / 216

页数：5

共 21 条

[1] COFACTOR AND SUBSTRATE REQUIREMENTS OF COLLAGEN PROLINE HYDROXYLASE
HUTTON, JJ
TAPPEL, AL
UDENFRIEND, S
[J]. ARCHIVES OF BIOCHEMISTRY AND BIOPHYSICS, 1967, 118 (01) : 231 - +
[2] A RAPID ASSAY FOR COLLAGEN PROLINE HYDROXYLASE
HUTTON, JJ
TAPPEL, AL
UDENFRIEND, S
[J]. ANALYTICAL BIOCHEMISTRY, 1966, 16 (03) : 384 - +
[3] ON THE SIGNIFICANCE OF THE EXTRACTABLE COLLAGENS
JACKSON, DS
BENTLEY, JP
[J]. JOURNAL OF BIOPHYSICAL AND BIOCHEMICAL CYTOLOGY, 1960, 7 (01): : 37 - 42
[4] MYOCARDIAL CONNECTIVE TISSUE METABOLISM IN RESPONSE TO INJURY - HISTOLOGICAL AND CHEMICAL STUDIES OF MULCOPOLYSACCHARIDE AND COLLAGEN IN RAT HEARTS AFTER ISOPROTERENOL-INDUCED INFARCTION
JUDD, JT
WEXLER, BC
[J]. CIRCULATION RESEARCH, 1969, 25 (02) : 201 - &
[5] MYOCARDIAL CONNECTIVE TISSUE METABOLISM IN RESPONSE TO INJURY 2. INVESTIGATION OF MUCOPOLYSACCHARIDES INVOLVED IN ISOPROTERENOL-INDUCED NECROSIS AND REPAIR IN RAT HEARTS
JUDD, JT
WEXLER, BC
[J]. CIRCULATION RESEARCH, 1970, 26 (01) : 101 - &
[6] S-35 UPTAKE IN ACID MUCOPOLYSACCHARIDES OF RAT-HEART FOLLOWING INJURY
JUDD, JT
WEXLER, BC
[J]. AMERICAN JOURNAL OF PHYSIOLOGY, 1973, 224 (02): : 312 - 317
[7] MYOCARDIAL GLYCOPROTEIN CHANGES WITH ISOPROTERENOL-INDUCED NECROSIS AND REPAIR IN RAT
JUDD, JT
WEXLER, BC
[J]. AMERICAN JOURNAL OF PHYSIOLOGY, 1974, 226 (03): : 597 - 602
[8] JUDD JT, 1969, P SOC EXP BIOL MED, V130, P1302
[9] PROTOCOLLAGEN PROLINE HYDROXYLASE-ACTIVITY IN RAT-HEART DURING EXPERIMENTAL CARDIAC HYPERTROPHY
LINDY, S
TURTO, H
UITTO, J
[J]. CIRCULATION RESEARCH, 1972, 30 (02) : 205 - +
[10] LOWRY OH, 1951, J BIOL CHEM, V193, P265

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