AMYOTROPHIC-LATERAL-SCLEROSIS - HUMAN CHALLENGE FOR NEUROSCIENCE

[1] FAMILIAL AMYOTROPHIC-LATERAL-SCLEROSIS (ALS) IN JAPAN ASSOCIATED WITH H46R MUTATION IN CU/ZN SUPEROXIDE-DISMUTASE GENE - A POSSIBLE NEW SUBTYPE OF FAMILIAL ALS [J]. JOURNAL OF THE NEUROLOGICAL SCIENCES, 1994, 126 (01) : 77 - 83

[2] ALS, SOD AND PEROXYNITRITE [J]. NATURE, 1993, 364 (6438) : 584 - 584

[3] NEUROFILAMENT LIGHT AND POLYADENYLATED MESSENGER-RNA LEVELS ARE DECREASED IN AMYOTROPHIC-LATERAL-SCLEROSIS MOTOR-NEURONS [J]. JOURNAL OF NEUROPATHOLOGY AND EXPERIMENTAL NEUROLOGY, 1994, 53 (03) : 221 - 230

[4] SUPEROXIDE-DISMUTASE-1 WITH MUTATIONS LINKED TO FAMILIAL AMYOTROPHIC-LATERAL-SCLEROSIS POSSESSES SIGNIFICANT ACTIVITY [J]. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA, 1994, 91 (17) : 8292 - 8296

[5] BORCHELT DR, 1995, IN PRESS J BIOL CHEM

[6] SUPEROXIDE-DISMUTASE ACTIVITY, OXIDATIVE DAMAGE, AND MITOCHONDRIAL ENERGY-METABOLISM IN FAMILIAL AND SPORADIC AMYOTROPHIC-LATERAL-SCLEROSIS [J]. JOURNAL OF NEUROCHEMISTRY, 1993, 61 (06) : 2322 - 2325

[7] BRACCO F, 1991, P SOC EXP BIOL MED, V196, P36

[8] CLINICAL IMPLICATIONS OF BASIC RESEARCH - A TRANSGENIC-MOUSE MODEL OF AMYOTROPHIC-LATERAL-SCLEROSIS [J]. NEW ENGLAND JOURNAL OF MEDICINE, 1994, 331 (16) : 1091 - 1092

[9] PROGRESSIVE NEURONOPATHY IN TRANSGENIC MICE EXPRESSING THE HUMAN NEUROFILAMENT HEAVY GENE - A MOUSE MODEL OF AMYOTROPHIC-LATERAL-SCLEROSIS [J]. CELL, 1993, 73 (01) : 35 - 46

[10] DALCANTO MC, 1994, AM J PATHOL, V145, P1271

[1] FAMILIAL AMYOTROPHIC-LATERAL-SCLEROSIS (ALS) IN JAPAN ASSOCIATED WITH H46R MUTATION IN CU/ZN SUPEROXIDE-DISMUTASE GENE - A POSSIBLE NEW SUBTYPE OF FAMILIAL ALS [J]. JOURNAL OF THE NEUROLOGICAL SCIENCES, 1994, 126 (01) : 77 - 83

[2] ALS, SOD AND PEROXYNITRITE [J]. NATURE, 1993, 364 (6438) : 584 - 584

[3] NEUROFILAMENT LIGHT AND POLYADENYLATED MESSENGER-RNA LEVELS ARE DECREASED IN AMYOTROPHIC-LATERAL-SCLEROSIS MOTOR-NEURONS [J]. JOURNAL OF NEUROPATHOLOGY AND EXPERIMENTAL NEUROLOGY, 1994, 53 (03) : 221 - 230

[4] SUPEROXIDE-DISMUTASE-1 WITH MUTATIONS LINKED TO FAMILIAL AMYOTROPHIC-LATERAL-SCLEROSIS POSSESSES SIGNIFICANT ACTIVITY [J]. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA, 1994, 91 (17) : 8292 - 8296

[5] BORCHELT DR, 1995, IN PRESS J BIOL CHEM

[6] SUPEROXIDE-DISMUTASE ACTIVITY, OXIDATIVE DAMAGE, AND MITOCHONDRIAL ENERGY-METABOLISM IN FAMILIAL AND SPORADIC AMYOTROPHIC-LATERAL-SCLEROSIS [J]. JOURNAL OF NEUROCHEMISTRY, 1993, 61 (06) : 2322 - 2325

[7] BRACCO F, 1991, P SOC EXP BIOL MED, V196, P36

[8] CLINICAL IMPLICATIONS OF BASIC RESEARCH - A TRANSGENIC-MOUSE MODEL OF AMYOTROPHIC-LATERAL-SCLEROSIS [J]. NEW ENGLAND JOURNAL OF MEDICINE, 1994, 331 (16) : 1091 - 1092

[9] PROGRESSIVE NEURONOPATHY IN TRANSGENIC MICE EXPRESSING THE HUMAN NEUROFILAMENT HEAVY GENE - A MOUSE MODEL OF AMYOTROPHIC-LATERAL-SCLEROSIS [J]. CELL, 1993, 73 (01) : 35 - 46

[10] DALCANTO MC, 1994, AM J PATHOL, V145, P1271