GLUCOSE AND THE METABOLISM OF ISCHEMIC MYOCARDIUM

被引:31
作者
OPIE, LH
机构
[1] Heart Research Unit, University of Capetown Medical School
来源
LANCET | 1995年 / 345卷 / 8964期
关键词
D O I
10.1016/S0140-6736(95)91080-8
中图分类号
R5 [内科学];
学科分类号
1002 ; 100201 ;
摘要
[No abstract available]
引用
收藏
页码:1520 / 1521
页数:2
相关论文
共 7 条
[1]  
Opie, The glucose hypothesis: relation to acute myocardial ischemia, J Mol Cell Cardiol, 1, pp. 107-115, (1970)
[2]  
Weiss, Lamp, Cardiac ATP-sensitive K<sup>+</sup>-channels. Evidence for preferential regulation by glycolysis, J Gen Physiol, 94, pp. 911-935, (1989)
[3]  
Owen, Dennis, Opie, Glucose flux rate regulates onset of ischemic contracture in globally underperfused rat hearts, Circ Res, 66, pp. 344-354, (1990)
[4]  
Eberli, Weinberg, Grice, Horowtiz, Apstein, Protective effect of increased glycolytic substrate against systolic and diastolic dysfunction and increased coronary resistance from prolonged global underperfusion and reperfusion in isolated rabbit hearts perfused with erythrocyte suspensions, Circ Res, 68, pp. 466-481, (1991)
[5]  
Neely, Grotyohann, Role of glycolytic products in damage to ischemic myocardium. Dissociation of adenosine triphosphate levels and recovery of function of reperfused ischemic hearts, Circ Res, 55, pp. 816-824, (1984)
[6]  
King, Boucher, Opie, Coronary flow and glucose delivery as determinants of contracture in the ischemic myocardium, J Mol Cell Cardiol, 27, pp. 701-720, (1995)
[7]  
Oliver, Opie, Effects of glucose and fatty acids on myocardial ischaemia and arrhythmias, Lancet, 343, pp. 155-158, (1994)