THE P53 TUMOR SUPPRESSOR PROTEIN - MEETING REVIEW

被引：144

作者：

PRIVES, C

MANFREDI, JJ

机构：

[1] Department of Biological Sciences, Columbia University, New York

来源：

GENES & DEVELOPMENT | 1993年 / 7卷 / 04期

关键词：

D O I：

10.1101/gad.7.4.529

中图分类号：

Q2 [细胞生物学];

学科分类号：

071009 ; 090102 ;

摘要：

[No abstract available]

引用

页码：529 / 534

页数：6

共 8 条

[1] MICE DEFICIENT FOR P53 ARE DEVELOPMENTALLY NORMAL BUT SUSCEPTIBLE TO SPONTANEOUS TUMORS [J].

DONEHOWER, LA ;

HARVEY, M ;

SLAGLE, BL ;

MCARTHUR, MJ ;

MONTGOMERY, CA ;

BUTEL, JS ;

BRADLEY, A .

NATURE, 1992, 356 (6366) :215-221

[2] DEFINITION OF A CONSENSUS BINDING-SITE FOR P53 [J].

ELDEIRY, WS ;

KERN, SE ;

PIETENPOL, JA ;

KINZLER, KW ;

VOGELSTEIN, B .

NATURE GENETICS, 1992, 1 (01) :45-49

[3] MAMMALIAN GENES COORDINATELY REGULATED BY GROWTH ARREST SIGNALS AND DNA-DAMAGING AGENTS [J].

FORNACE, AJ ;

NEBERT, DW ;

HOLLANDER, MC ;

LUETHY, JD ;

PAPATHANASIOU, M ;

FARGNOLI, J ;

HOLBROOK, NJ .

MOLECULAR AND CELLULAR BIOLOGY, 1989, 9 (10) :4196-4203

[4] A TRANSCRIPTIONALLY ACTIVE DNA-BINDING SITE FOR HUMAN P53 PROTEIN COMPLEXES [J].

FUNK, WD ;

PAK, DT ;

KARAS, RH ;

WRIGHT, WE ;

SHAY, JW .

MOLECULAR AND CELLULAR BIOLOGY, 1992, 12 (06) :2866-2871

[5] WILD-TYPE P53 IS A CELL-CYCLE CHECKPOINT DETERMINANT FOLLOWING IRRADIATION [J].

KUERBITZ, SJ ;

PLUNKETT, BS ;

WALSH, WV ;

KASTAN, MB .

PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA, 1992, 89 (16) :7491-7495

[6] THE MDM-2 ONCOGENE PRODUCT FORMS A COMPLEX WITH THE P53 PROTEIN AND INHIBITS P53-MEDIATED TRANSACTIVATION [J].

MOMAND, J ;

ZAMBETTI, GP ;

OLSON, DC ;

GEORGE, D ;

LEVINE, AJ .

CELL, 1992, 69 (07) :1237-1245

[7]

STURZBECHER HW, 1992, ONCOGENE, V7, P1513

[8] WILD-TYPE P53 INDUCES APOPTOSIS OF MYELOID LEUKEMIC-CELLS THAT IS INHIBITED BY INTERLEUKIN-6 [J].

YONISHROUACH, E ;

RESNITZKY, D ;

LOTEM, J ;

SACHS, L ;

KIMCHI, A ;

OREN, M .

NATURE, 1991, 352 (6333) :345-347

← 1 →