STUDIES ON THE MECHANISM OF ACTION OF ISONIAZID AND ETHIONAMIDE IN THE CHEMOTHERAPY OF TUBERCULOSIS

被引：197

作者：

JOHNSSON, K

KING, DS

SCHULTZ, PG

机构：

[1] UNIV CALIF BERKELEY, HOWARD HUGHES MED INST, DEPT CHEM, BERKELEY, CA 94720 USA

[2] UNIV CALIF BERKELEY, HOWARD HUGHES MED INST, DEPT MOLEC & CELL BIOL, BERKELEY, CA 94720 USA

来源：

JOURNAL OF THE AMERICAN CHEMICAL SOCIETY | 1995年 / 117卷 / 17期

关键词：

D O I：

10.1021/ja00122a038

中图分类号：

O6 [化学];

学科分类号：

0703 ;

摘要：

引用

页码：5009 / 5010

页数：2

共 13 条

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[8] MANGANESE-MEDIATED OXIDATIVE DAMAGE OF CELLULAR AND ISOLATED DNA BY ISONIAZID AND RELATED HYDRAZINES - NON-FENTON-TYPE HYDROXYL RADICAL FORMATION
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[9] MECHANISTIC STUDIES OF THE OXIDATION OF ISONIAZID BY THE CATALASE PEROXIDASE FROM MYCOBACTERIUM-TUBERCULOSIS
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[J]. JOURNAL OF THE AMERICAN CHEMICAL SOCIETY, 1994, 116 (16) : 7425 - 7426
[10] EVIDENCE FOR THE GENERATION OF ACTIVE OXYGEN BY ISONIAZID TREATMENT OF EXTRACTS OF MYCOBACTERIUM-TUBERCULOSIS H37RA
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