POSSIBLE PATHWAY FOR FORMATION OF FIBRIN DEGRADATION PRODUCTS DURING ANCROD THERAPY

被引：14

作者：

MCKILLOP, C ^{[1
]}

EDGAR, W ^{[1
]}

FORBES, CD ^{[1
]}

PRENTICE, CRM ^{[1
]}

机构：

[1] UNIV GLASGOW, ROY INFIRM, DEPT MED, GLASGOW G4 0SF, SCOTLAND

来源：

NATURE | 1975年 / 255卷 / 5510期

关键词：

D O I：

10.1038/255638a0

中图分类号：

O [数理科学和化学]; P [天文学、地球科学]; Q [生物科学]; N [自然科学总论];

学科分类号：

07 ; 0710 ; 09 ;

摘要：

引用

页码：638 / 639

页数：2

共 12 条

[1]

ASBECK F, IN PRESS

[2]

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[3]

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[4] MODE OF ACTION OF ANCROD AS A DEFIBRINATING AGENT [J].

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[5] CHARACTERIZATION OF PEPTIDES RELEASED FROM HUMAN FIBRINOGEN BY ARVIN [J].

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COEN, LJ .

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[6] FIBRIN-I DEGRADATION PRODUCTS WITH A MOLECULAR-WEIGHT HIGHER THAN THAT OF FIBRINOGEN - IDENTIFICATION AND CHARACTERIZATION OF IN-VITRO PRODUCTS FROM HUMAN PLASMA [J].

HUGO, RV ;

GRAEFF, H .

THROMBOSIS ET DIATHESIS HAEMORRHAGICA, 1973, 29 (01) :122-129

[7]

KONTTINEN YP, 1973, THROMB DIATH HAEMOST, V30, P403

[8]

Mancini G, 1965, Immunochemistry, V2, P235, DOI 10.1016/0019-2791(65)90004-2

[9] DIFFERENCES IN SUBUNIT STRUCTURE OF HUMAN FIBRIN FORMED BY ACTION OFARVIN, REPTILASE AND THROMBIN [J].

MATTOCK, P ;

ESNOUF, MP .

NATURE-NEW BIOLOGY, 1971, 233 (43) :277-+

[10] BLOOD FIBRINOLYTIC ACTIVITY DURING ARVIN THERAPY [J].

PITNEY, WR ;

BELL, WR ;

BOLTON, G .

BRITISH JOURNAL OF HAEMATOLOGY, 1969, 16 (1-2) :165-&

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