THE MECHANISM OF SKIN TUMOR PROMOTION CAUSED BY PHORBOL ESTERS - POSSIBLE INVOLVEMENT OF ARACHIDONIC-ACID CASCADE LIPOXYGENASE, PROTEIN KINASE-C AND CALCIUM CALMODULIN SYSTEMS

被引：54

作者：

NAKADATE, T

机构：

来源：

JAPANESE JOURNAL OF PHARMACOLOGY | 1989年 / 49卷 / 01期

关键词：

D O I：

10.1254/jjp.49.1

中图分类号：

R9 [药学];

学科分类号：

1007 ;

摘要：

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页码：1 / 9

页数：9

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[1] PALMITOYLCARNITINE REVERSES 12-O-TETRADECANOYLPHORBOL-13-ACETATE-INDUCED REFRACTORY STATE FOR THE TPA-CAUSED ORNITHINE DECARBOXYLASE INDUCTION IN MOUSE EPIDERMIS
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[2] INHIBITION OF 12-O-TETRADECANOYLPHORBOL-13-ACETATE-MEDIATED EPIDERMAL ORNITHINE DECARBOXYLASE INDUCTION AND SKIN TUMOR PROMOTION BY NEW LIPOXYGENASE INHIBITORS LACKING PROTEIN-KINASE-C INHIBITORY EFFECTS
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[3] BEDFORD CJ, 1983, J INVEST DERMATOL, V81, P566
[4] PHORBOL ESTERS AS PROBES OF THE MODULATORY SITE ON PROTEIN-KINASE-C - AN OVERVIEW
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[5] BLUMBERG PM, 1983, MECHANISMS TUMOR PRO, P143
[6] PHOSPHOLIPASE-ACTIVITY IN SKIN AFTER APPLICATION OF PHORBOL ESTERS AND 3-METHYLCHOLANTHRENE
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[J]. CARCINOGENESIS, 1981, 2 (11) : 1119 - 1122
[7] Fischer S.M., 1985, ARACHIDONIC ACID MET, P21
[8] INDOMETHACIN ENHANCEMENT OF TPA TUMOR PROMOTION IN MICE
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[J]. CANCER LETTERS, 1980, 10 (04) : 343 - 350
[9] INHIBITION OF MOUSE SKIN TUMOR PROMOTION BY SEVERAL INHIBITORS OF ARACHIDONIC-ACID METABOLISM
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