RAS P21 PROTEINS WITH HIGH OR LOW GTPASE ACTIVITY CAN EFFICIENTLY TRANSFORM NIH/3T3 CELLS

[1] STRUCTURAL ORGANIZATION AND BIOLOGICAL-ACTIVITY OF MOLECULAR CLONES OF THE INTEGRATED GENOME OF A BALB-C MOUSE SARCOMA-VIRUS [J]. JOURNAL OF VIROLOGY, 1981, 40 (02) : 431 - 439

[2] GENERATION OF BALB-MUSV AND HA-MUSV BY TYPE-C VIRUS TRANSDUCTION OF HOMOLOGOUS TRANSFORMING GENES FROM DIFFERENT SPECIES [J]. CELL, 1981, 26 (01) : 129 - 134

[3] BRADFORD MM, 1976, ANAL BIOCHEM, V72, P248, DOI 10.1016/0003-2697(76)90527-3

[4] ACTIVATION OF KI-RAS 2 GENE IN HUMAN-COLON AND LUNG CARCINOMAS BY 2 DIFFERENT POINT MUTATIONS [J]. NATURE, 1983, 304 (5926) : 507 - 513

[5] TUMORIGENIC TRANSFORMATION OF MAMMALIAN-CELLS INDUCED BY A NORMAL HUMAN-GENE HOMOLOGOUS TO THE ONCOGENE OF HARVEY MURINE SARCOMA-VIRUS [J]. NATURE, 1982, 297 (5866) : 479 - 483

[6] THE P21 SRC GENES OF HARVEY AND KIRSTEN SARCOMA-VIRUSES ORIGINATE FROM DIVERGENT MEMBERS OF A FAMILY OF NORMAL VERTEBRATE GENES [J]. NATURE, 1981, 292 (5823) : 506 - 511

[7] ANALYSIS OF THE TRANSFORMING POTENTIAL OF THE HUMAN H-RAS GENE BY RANDOM MUTAGENESIS [J]. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA-BIOLOGICAL SCIENCES, 1984, 81 (13): : 4008 - 4012

[8] MICROINJECTION OF THE ONCOGENE FORM OF THE HUMAN H-RAS (T-24) PROTEIN RESULTS IN RAPID PROLIFERATION OF QUIESCENT CELLS [J]. CELL, 1984, 38 (01) : 109 - 117

[9] ACTIVATION OF RAS GENES IN HUMAN-TUMORS DOES NOT AFFECT LOCALIZATION, MODIFICATION, OR NUCLEOTIDE BINDING-PROPERTIES OF P21 [J]. CELL, 1984, 37 (01) : 151 - 158

[10] MONOCLONAL-ANTIBODIES TO THE P21 PRODUCTS OF THE TRANSFORMING GENE OF HARVEY MURINE SARCOMA-VIRUS AND OF THE CELLULAR RAS GENE FAMILY [J]. JOURNAL OF VIROLOGY, 1982, 43 (01) : 294 - 304

[1] STRUCTURAL ORGANIZATION AND BIOLOGICAL-ACTIVITY OF MOLECULAR CLONES OF THE INTEGRATED GENOME OF A BALB-C MOUSE SARCOMA-VIRUS [J]. JOURNAL OF VIROLOGY, 1981, 40 (02) : 431 - 439

[2] GENERATION OF BALB-MUSV AND HA-MUSV BY TYPE-C VIRUS TRANSDUCTION OF HOMOLOGOUS TRANSFORMING GENES FROM DIFFERENT SPECIES [J]. CELL, 1981, 26 (01) : 129 - 134

[3] BRADFORD MM, 1976, ANAL BIOCHEM, V72, P248, DOI 10.1016/0003-2697(76)90527-3

[4] ACTIVATION OF KI-RAS 2 GENE IN HUMAN-COLON AND LUNG CARCINOMAS BY 2 DIFFERENT POINT MUTATIONS [J]. NATURE, 1983, 304 (5926) : 507 - 513

[5] TUMORIGENIC TRANSFORMATION OF MAMMALIAN-CELLS INDUCED BY A NORMAL HUMAN-GENE HOMOLOGOUS TO THE ONCOGENE OF HARVEY MURINE SARCOMA-VIRUS [J]. NATURE, 1982, 297 (5866) : 479 - 483

[6] THE P21 SRC GENES OF HARVEY AND KIRSTEN SARCOMA-VIRUSES ORIGINATE FROM DIVERGENT MEMBERS OF A FAMILY OF NORMAL VERTEBRATE GENES [J]. NATURE, 1981, 292 (5823) : 506 - 511

[7] ANALYSIS OF THE TRANSFORMING POTENTIAL OF THE HUMAN H-RAS GENE BY RANDOM MUTAGENESIS [J]. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA-BIOLOGICAL SCIENCES, 1984, 81 (13): : 4008 - 4012

[8] MICROINJECTION OF THE ONCOGENE FORM OF THE HUMAN H-RAS (T-24) PROTEIN RESULTS IN RAPID PROLIFERATION OF QUIESCENT CELLS [J]. CELL, 1984, 38 (01) : 109 - 117

[9] ACTIVATION OF RAS GENES IN HUMAN-TUMORS DOES NOT AFFECT LOCALIZATION, MODIFICATION, OR NUCLEOTIDE BINDING-PROPERTIES OF P21 [J]. CELL, 1984, 37 (01) : 151 - 158

[10] MONOCLONAL-ANTIBODIES TO THE P21 PRODUCTS OF THE TRANSFORMING GENE OF HARVEY MURINE SARCOMA-VIRUS AND OF THE CELLULAR RAS GENE FAMILY [J]. JOURNAL OF VIROLOGY, 1982, 43 (01) : 294 - 304