HUMAN KERATINOCYTES SYNTHESIZE AND SECRETE THE EXTRACELLULAR-MATRIX PROTEIN, THROMBOSPONDIN

被引：43

作者：

WIKNER, NE

DIXIT, VM

FRAZIER, WA

CLARK, RAF

机构：

[1] NATL JEWISH CTR IMMUNOL & RESP MED,DEPT MED,1400 JACKSON ST,DENVER,CO 80206

[2] UNIV COLORADO,SCH MED,DEPT DERMATOL,DENVER,CO 80202

[3] WASHINGTON UNIV,DEPT BIOL CHEM,ST LOUIS,MO 63130

来源：

JOURNAL OF INVESTIGATIVE DERMATOLOGY | 1987年 / 88卷 / 02期

关键词：

D O I：

10.1111/1523-1747.ep12525350

中图分类号：

R75 [皮肤病学与性病学];

学科分类号：

100206 ;

摘要：

引用

页码：207 / 211

页数：5

共 33 条

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[2] CALCIUM-REGULATED DIFFERENTIATION OF NORMAL HUMAN EPIDERMAL-KERATINOCYTES IN CHEMICALLY DEFINED CLONAL CULTURE AND SERUM-FREE SERIAL CULTURE
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HAM, RG
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[3] HUMAN KERATINOCYTES THAT HAVE NOT TERMINALLY DIFFERENTIATED SYNTHESIZE LAMININ AND FIBRONECTIN BUT DEPOSIT ONLY FIBRONECTIN IN THE PERICELLULAR MATRIX
CLARK, RAF
NIELSEN, LD
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[J]. JOURNAL OF CELLULAR BIOCHEMISTRY, 1985, 28 (02) : 127 - 141
[4] COLIGAN JE, 1984, J BIOL CHEM, V259, P3944
[5] FORMATION OF A STABLE COMPLEX OF THROMBIN AND THE SECRETED PLATELET PROTEIN GLYCOPROTEIN-G (THROMBIN-SENSITIVE PROTEIN, THROMBOSPONDIN) BY THIOL DISULFIDE EXCHANGE
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[J]. BIOCHEMISTRY, 1984, 23 (21) : 4984 - 4990
[6] DIXIT VM, 1986, J BIOL CHEM, V261, P1962
[7] ISOLATION OF THE FIBRINOGEN-BINDING REGION OF PLATELET THROMBOSPONDIN
DIXIT, VM
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SANTORO, SA
[J]. BIOCHEMICAL AND BIOPHYSICAL RESEARCH COMMUNICATIONS, 1984, 119 (03) : 1075 - 1081
[8] DIXIT VM, 1984, J BIOL CHEM, V259, P100
[9] A MONOCLONAL-ANTIBODY AGAINST HUMAN THROMBOSPONDIN INHIBITS PLATELET-AGGREGATION
DIXIT, VM
HAVERSTICK, DM
OROURKE, KM
HENNESSY, SW
GRANT, GA
SANTORO, SA
FRAZIER, WA
[J]. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA, 1985, 82 (10) : 3472 - 3476
[10] INHIBITION OF PLATELET-AGGREGATION BY A MONOCLONAL-ANTIBODY AGAINST HUMAN FIBRONECTIN
DIXIT, VM
HAVERSTICK, DM
OROURKE, K
HENNESSY, SW
BROEKELMANN, TJ
MCDONALD, JA
GRANT, GA
SANTORO, SA
FRAZIER, WA
[J]. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA, 1985, 82 (11) : 3844 - 3848

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