PROLONGED RESPONSIVENESS TO OUABAIN IN HYPERTROPHIED RAT-HEART - PHYSIOLOGICAL AND BIOCHEMICAL-EVIDENCE

[1] MODULATION OF CARDIAC GLYCOSIDE INHIBITION OF (NA++K+)-ATPASE BY MEMBRANE-LIPIDS - DIFFERENCE BETWEEN SPECIES [J]. BIOCHIMICA ET BIOPHYSICA ACTA, 1983, 729 (01) : 75 - 84

[2] HIGH-AFFINITY OUABAIN BINDING-SITE AND LOW-DOSE POSITIVE INOTROPIC EFFECT IN RAT MYOCARDIUM [J]. NATURE, 1982, 296 (5853) : 167 - 169

[3] MYOCARDIAL MEMBRANES - REGULATION AND FUNCTION OF THE SODIUM-PUMP [J]. ANNUAL REVIEW OF PHYSIOLOGY, 1982, 44 : 375 - 388

[4] INCREASED MYOTHERMAL ECONOMY OF ISOMETRIC FORCE GENERATION IN COMPENSATED CARDIAC-HYPERTROPHY INDUCED BY PULMONARY-ARTERY CONSTRICTION IN THE RABBIT - A CHARACTERIZATION OF HEAT LIBERATION IN NORMAL AND HYPERTROPHIED RIGHT VENTRICULAR PAPILLARY-MUSCLES [J]. CIRCULATION RESEARCH, 1982, 50 (04) : 491 - 500

[5] VENTRICULAR CONTRACTURE AND COMPLIANCE CHANGES WITH GLOBAL ISCHEMIA AND REPERFUSION, AND THEIR EFFECT ON CORONARY RESISTANCE IN RAT [J]. CIRCULATION RESEARCH, 1977, 41 (02) : 206 - 217

[6] ARONSON RS, 1983, MYOCARDIAL HYPERTROP, P233

[7] DECREASED CATECHOLAMINE SENSITIVITY AND BETA-ADRENERGIC-RECEPTOR DENSITY IN FAILING HUMAN HEARTS [J]. NEW ENGLAND JOURNAL OF MEDICINE, 1982, 307 (04) : 205 - 211

[8] CHARLEMAGNE D, 1986, J BIOL CHEM, V261, P185

[9] CUTILLETTA AF, 1975, J MOL CELL CARDIOL, V7, P767, DOI 10.1016/0022-2828(75)90042-5

[10] CARDIAC GLYCOSIDE RECEPTOR, (NA+ + K+)-ATPASE ACTIVITY AND FORCE OF CONTRACTION IN RAT-HEART [J]. BIOCHEMICAL PHARMACOLOGY, 1980, 29 (24) : 3219 - 3229

[1] MODULATION OF CARDIAC GLYCOSIDE INHIBITION OF (NA++K+)-ATPASE BY MEMBRANE-LIPIDS - DIFFERENCE BETWEEN SPECIES [J]. BIOCHIMICA ET BIOPHYSICA ACTA, 1983, 729 (01) : 75 - 84

[2] HIGH-AFFINITY OUABAIN BINDING-SITE AND LOW-DOSE POSITIVE INOTROPIC EFFECT IN RAT MYOCARDIUM [J]. NATURE, 1982, 296 (5853) : 167 - 169

[3] MYOCARDIAL MEMBRANES - REGULATION AND FUNCTION OF THE SODIUM-PUMP [J]. ANNUAL REVIEW OF PHYSIOLOGY, 1982, 44 : 375 - 388

[4] INCREASED MYOTHERMAL ECONOMY OF ISOMETRIC FORCE GENERATION IN COMPENSATED CARDIAC-HYPERTROPHY INDUCED BY PULMONARY-ARTERY CONSTRICTION IN THE RABBIT - A CHARACTERIZATION OF HEAT LIBERATION IN NORMAL AND HYPERTROPHIED RIGHT VENTRICULAR PAPILLARY-MUSCLES [J]. CIRCULATION RESEARCH, 1982, 50 (04) : 491 - 500

[5] VENTRICULAR CONTRACTURE AND COMPLIANCE CHANGES WITH GLOBAL ISCHEMIA AND REPERFUSION, AND THEIR EFFECT ON CORONARY RESISTANCE IN RAT [J]. CIRCULATION RESEARCH, 1977, 41 (02) : 206 - 217

[6] ARONSON RS, 1983, MYOCARDIAL HYPERTROP, P233

[7] DECREASED CATECHOLAMINE SENSITIVITY AND BETA-ADRENERGIC-RECEPTOR DENSITY IN FAILING HUMAN HEARTS [J]. NEW ENGLAND JOURNAL OF MEDICINE, 1982, 307 (04) : 205 - 211

[8] CHARLEMAGNE D, 1986, J BIOL CHEM, V261, P185

[9] CUTILLETTA AF, 1975, J MOL CELL CARDIOL, V7, P767, DOI 10.1016/0022-2828(75)90042-5

[10] CARDIAC GLYCOSIDE RECEPTOR, (NA+ + K+)-ATPASE ACTIVITY AND FORCE OF CONTRACTION IN RAT-HEART [J]. BIOCHEMICAL PHARMACOLOGY, 1980, 29 (24) : 3219 - 3229