PATHOGENESIS OF COAGULATION DEFECT DEVELOPING DURING PATHOLOGICAL PLASMA PROTEOLYTIC (FIBRINOLYTIC) STATES .1. SIGNIFICANCE OF FIBRINOGEN PROTEOLYSIS AND CIRCULATING FIBRINOGEN BREAKDOWN PRODUCTS

被引:268
作者
FLETCHER, AP
SHERRY, S
ALKJAERSIG, N
机构
关键词
D O I
10.1172/JCI104546
中图分类号
R-3 [医学研究方法]; R3 [基础医学];
学科分类号
1001 ;
摘要
引用
收藏
页码:896 / &
相关论文
共 73 条
[1]   INHIBITION OF PLASMIN, TRYPSIN AND THE STREPTOKINASE-ACTIVATED FIBRINOLYTIC SYSTEM BY EPSILON-AMINOCAPROIC ACID [J].
ABLONDI, FB ;
HAGAN, JJ ;
PHILIPS, M ;
DERENZO, EC .
ARCHIVES OF BIOCHEMISTRY AND BIOPHYSICS, 1959, 82 (01) :153-160
[2]  
ABOULKER P, 1955, PRESSE MED, V63, P353
[3]  
ALAGILLE D, 1956, Sem Hop, V32, P355
[4]  
ALKJAERSIG N, 1958, J BIOL CHEM, V233, P81
[5]  
ALKJAERSIG N, 1959, J BIOL CHEM, V234, P832
[6]   PATHOGENESIS OF COAGULATION DEFECT DEVELOPING DURING PATHOLOGICAL PLASMA PROTEOLYTIC (FIBRINOLYTIC) STATES .2. SIGNIFICANCE, MECHANISM AND CONSEQUENCES OF DEFECTIVE FIBRIN POLYMERIZATION [J].
ALKJAERSIG, N ;
SHERRY, S ;
FLETCHER, AP .
JOURNAL OF CLINICAL INVESTIGATION, 1962, 41 (04) :917-&
[7]   MECHANISM OF CLOT DISSOLUTION BY PLASMIN [J].
ALKJAERSIG, N ;
FLETCHER, AP ;
SHERRY, S .
JOURNAL OF CLINICAL INVESTIGATION, 1959, 38 (07) :1086-1095
[8]  
ALKJAERSIG N, UNPUBLISHED
[9]  
ALKJAERSIG N, 1961, HENRY FORD HOSPITAL
[10]  
BEAUMONT JL, 1956, REV FR ETUD CLIN BIO, V1, P667