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TYROSINE PHOSPHORYLATION AND CA2+ INFLUX

被引：9
作者：
SAGE, SO
SARGEANT, P
JENNER, S
FARNDALE, RW
机构：
[1] MAYO CLIN & MAYO FDN,DEPT PHARMACOL,ROCHESTER,MN 55905
[2] UNIV CAMBRIDGE,DEPT BIOCHEM,CAMBRIDGE CB2 1QW,ENGLAND
来源：
TRENDS IN PHARMACOLOGICAL SCIENCES | 1994年 / 15卷 / 08期
关键词：
D O I：
10.1016/0165-6147(94)90007-8
中图分类号：
R9 [药学];
学科分类号：
1007 ;
摘要：
引用
收藏
页码：282 / 282
页数：1
相关论文
共 6 条
[1]   RECEPTOR-ACTIVATED CA2+ INFLUX - HOW MANY MECHANISMS FOR HOW MANY CHANNELS [J].
FASOLATO, C ;
INNOCENTI, B ;
POZZAN, T .
TRENDS IN PHARMACOLOGICAL SCIENCES, 1994, 15 (03) :77-83
[2]  
LEE KM, 1993, J BIOL CHEM, V268, P9945
[3]   THE TYROSINE KINASE INHIBITORS METHYL 2,5-DIHYDROXYCINNAMATE AND GENISTEIN REDUCE THROMBIN-EVOKED TYROSINE PHOSPHORYLATION AND CA-2+ ENTRY IN HUMAN PLATELETS [J].
SARGEANT, P ;
FARNDALE, RW ;
SAGE, SO .
FEBS LETTERS, 1993, 315 (03) :242-246
[4]   CALCIUM STORE DEPLETION IN DIMETHYL BAPTA-LOADED HUMAN PLATELETS INCREASES PROTEIN-TYROSINE PHOSPHORYLATION IN THE ABSENCE OF A RISE IN CYTOSOLIC CALCIUM [J].
SARGEANT, P ;
FARNDALE, RW ;
SAGE, SO .
EXPERIMENTAL PHYSIOLOGY, 1994, 79 (02) :269-272
[5]  
SARGEANT P, 1993, J BIOL CHEM, V268, P18151
[6]  
VOSTAL JG, 1991, J BIOL CHEM, V266, P16911
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