LACK OF CHANGE IN HIGH-AFFINITY GLUTAMATE UPTAKE IN NUCLEUS TRACTUS SOLITARIUS FOLLOWING REMOVAL OF THE NODOSE GANGLION

被引：19

作者：

SVED, AF ^{[1
]}

机构：

[1] UNIV MED & DENT NEW JERSEY,DEPT NEUROSCI,NEWARK,NJ 07103

来源：

BRAIN RESEARCH BULLETIN | 1986年 / 16卷 / 03期

关键词：

D O I：

10.1016/0361-9230(86)90053-5

中图分类号：

Q189 [神经科学];

学科分类号：

071006 ;

摘要：

引用

页码：325 / 329

页数：5

共 19 条

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[2]

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[4] RELEASE OF [H-3]-LABELED L-GLUTAMINE ACID (L-GLU) AND [H-3]-LABELED D-ASPARTIC ACID (D-ASP) IN THE AREA OF NUCLEUS TRACTUS SOLITARIUS INVIVO PRODUCED BY STIMULATION OF THE VAGUS NERVE [J].

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REIS, DJ .

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[5] INVIVO RELEASE BY VAGAL-STIMULATION OF L-[H-3] GLUTAMIC-ACID IN THE NUCLEUS TRACTUS SOLITARIUS PRELOADED WITH L-[H-3] GLUTAMINE [J].

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[6]

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[7] HIGH AFFINITY UPTAKE SYSTEMS FOR GLYCINE GLUTAMIC AND ASPARTIC ACIDS IN SYNAPTOSOMES OF RAT CENTRAL NERVOUS TISSUES [J].

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[8]

LOWRY OH, 1951, J BIOL CHEM, V193, P265

[9] GLUTAMATERGIC CORTICOSTRIATAL PATH [J].

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[10] BIOCHEMICAL-EVIDENCE THAT L-GLUTAMATE IS A NEUROTRANSMITTER OF PRIMARY VAGAL AFFERENT NERVE-FIBERS [J].

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