DISTINGUISHING MINIMAL-CHANGE DISEASE FROM MESANGIAL DISORDERS

[1] A report of the Southwest Pediatric Nephrology Study Group, 1983, KIDNEY INT, V23, P87

[2] COMPLEMENT MEMBRANE ATTACK COMPLEX STIMULATES PRODUCTION OF REACTIVE OXYGEN METABOLITES BY CULTURED RAT MESANGIAL CELLS [J]. JOURNAL OF CLINICAL INVESTIGATION, 1986, 77 (03) : 762 - 767

[3] IMMUNE DEPOSITS AND MESANGIAL HYPERCELLULARITY IN MINIMAL CHANGE NEPHROTIC SYNDROME - CLINICAL RELEVANCE [J]. JOURNAL OF PEDIATRICS, 1982, 100 (02) : 188 - 191

[4] THERAPEUTIC ADVANTAGE OF CONVERTING ENZYME-INHIBITORS IN ARRESTING PROGRESSIVE RENAL-DISEASE ASSOCIATED WITH SYSTEMIC HYPERTENSION IN THE RAT [J]. JOURNAL OF CLINICAL INVESTIGATION, 1986, 77 (06) : 1993 - 2000

[5] CONTROL OF GLOMERULAR HYPERTENSION LIMITS GLOMERULAR INJURY IN RATS WITH REDUCED RENAL MASS [J]. JOURNAL OF CLINICAL INVESTIGATION, 1985, 76 (02) : 612 - 619

[6] [Anonymous], 1981, KIDNEY INT, V20, P765

[7] CONTRACTION OF CULTURED RAT GLOMERULAR CELLS OF APPARENT MESANGIAL ORIGIN AFTER STIMULATION WITH ANGIOTENSIN-II AND ARGININE VASOPRESSIN [J]. JOURNAL OF CLINICAL INVESTIGATION, 1980, 65 (03) : 754 - 760

[8] LOSS OF GLOMERULAR POLYANION INVITRO INDUCED BY MONONUCLEAR BLOOD-CELLS FROM PATIENTS WITH MINIMAL-CHANGE NEPHROTIC SYNDROME [J]. AMERICAN JOURNAL OF NEPHROLOGY, 1986, 6 (02) : 107 - 111

[9] CHRONIC GLOMERULONEPHRITIS - NON-IMMUNOLOGIC MECHANISMS OF PROGRESSIVE GLOMERULAR DAMAGE [J]. KIDNEY INTERNATIONAL, 1982, 21 (01) : 109 - 120

[10] BENACERRAF BARUJ, 1959, AMER JOUR PATHOL, V35, P75

[1] A report of the Southwest Pediatric Nephrology Study Group, 1983, KIDNEY INT, V23, P87

[2] COMPLEMENT MEMBRANE ATTACK COMPLEX STIMULATES PRODUCTION OF REACTIVE OXYGEN METABOLITES BY CULTURED RAT MESANGIAL CELLS [J]. JOURNAL OF CLINICAL INVESTIGATION, 1986, 77 (03) : 762 - 767

[3] IMMUNE DEPOSITS AND MESANGIAL HYPERCELLULARITY IN MINIMAL CHANGE NEPHROTIC SYNDROME - CLINICAL RELEVANCE [J]. JOURNAL OF PEDIATRICS, 1982, 100 (02) : 188 - 191

[4] THERAPEUTIC ADVANTAGE OF CONVERTING ENZYME-INHIBITORS IN ARRESTING PROGRESSIVE RENAL-DISEASE ASSOCIATED WITH SYSTEMIC HYPERTENSION IN THE RAT [J]. JOURNAL OF CLINICAL INVESTIGATION, 1986, 77 (06) : 1993 - 2000

[5] CONTROL OF GLOMERULAR HYPERTENSION LIMITS GLOMERULAR INJURY IN RATS WITH REDUCED RENAL MASS [J]. JOURNAL OF CLINICAL INVESTIGATION, 1985, 76 (02) : 612 - 619

[6] [Anonymous], 1981, KIDNEY INT, V20, P765

[7] CONTRACTION OF CULTURED RAT GLOMERULAR CELLS OF APPARENT MESANGIAL ORIGIN AFTER STIMULATION WITH ANGIOTENSIN-II AND ARGININE VASOPRESSIN [J]. JOURNAL OF CLINICAL INVESTIGATION, 1980, 65 (03) : 754 - 760

[8] LOSS OF GLOMERULAR POLYANION INVITRO INDUCED BY MONONUCLEAR BLOOD-CELLS FROM PATIENTS WITH MINIMAL-CHANGE NEPHROTIC SYNDROME [J]. AMERICAN JOURNAL OF NEPHROLOGY, 1986, 6 (02) : 107 - 111

[9] CHRONIC GLOMERULONEPHRITIS - NON-IMMUNOLOGIC MECHANISMS OF PROGRESSIVE GLOMERULAR DAMAGE [J]. KIDNEY INTERNATIONAL, 1982, 21 (01) : 109 - 120

[10] BENACERRAF BARUJ, 1959, AMER JOUR PATHOL, V35, P75