PROTECTION AGAINST IRON-INDUCED UROPORPHYRIA IN C57BL 10SCSN MICE BY THE PEROXISOME PROLIFERATOR NAFENOPIN

[1] Smith, De Matteis, Oxidative injury mediated by the hepatic cytochrome P-450 system in conjunction with cellular iron. Effects on the pathway of haem biosynthesis, Xenobiotica, 20, pp. 865-877, (1990)

[2] Sweeney, Jones, Cole, Basford, Krestynski, Iron deficiency prevents liver toxicity of 2,3,7,8-tetrachlorodibenzo-p-dioxin, Science, 204, pp. 332-335, (1979)

[3] Jones, Cole, Sweeney, The role of iron in the toxicity of 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD), Toxicol Appl Pharmacol, 61, pp. 74-88, (1981)

[4] Greig, Francis, Kay, Lovell, Smith, Incomplete correlation of 2,3,7,8-tetrachlor-odibenzo-p-dioxin hepatotoxicity with Ah phenotype in mice, Toxicol Appl Pharmacol, 74, pp. 17-25, (1984)

[5] Smith, Francis, Carthew, Iron as a synergist for hepatocellular carcinoma induced by polychlorinated biphenyls in Ah-responsive C57BL/10ScSn mice, Carcinogenesis, 11, pp. 437-444, (1990)

[6] Smith, Francis, Synergism of iron and hexachlorobenzene inhibits hepatic uroporphyrinogen decarboxylase in inbred mice with iron overload, Biochem J, 214, pp. 909-913, (1983)

[7] Rizzardini, Graziani, Carugo, Cantoni, Investigations on the role of free radical processes in hexachlorobenzene-induced porphyria in mice, J Biochem Toxicol, 3, pp. 33-46, (1988)

[8] Sweeney, Porphyria cutanea tarda, or the uroporphyrinogen decarboxylase deficiency diseases, Clin Biochem, 19, pp. 3-15, (1986)

[9] Francis, Smith, Polycyclic aromatic hydrocarbons cause hepatic porphyria in iron-loaded C57BL/ 10 mice: comparison of uroporphyrinogen decarboxylase inhibition with induction of alkoxyphenoxazone dealkylations, Biochem Biophys Res Commun, 146, pp. 13-20, (1987)

[10] Urquhart, Elder, Roberts, Lambrecht, Sinclair, Bement, Gorman, Sinclair, Uroporphyria produced in mice by 20-methylcholanthrene and 5-aminolevulinic acid, Biochem J, 253, pp. 357-362, (1988)

[1] Smith, De Matteis, Oxidative injury mediated by the hepatic cytochrome P-450 system in conjunction with cellular iron. Effects on the pathway of haem biosynthesis, Xenobiotica, 20, pp. 865-877, (1990)

[2] Sweeney, Jones, Cole, Basford, Krestynski, Iron deficiency prevents liver toxicity of 2,3,7,8-tetrachlorodibenzo-p-dioxin, Science, 204, pp. 332-335, (1979)

[3] Jones, Cole, Sweeney, The role of iron in the toxicity of 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD), Toxicol Appl Pharmacol, 61, pp. 74-88, (1981)

[4] Greig, Francis, Kay, Lovell, Smith, Incomplete correlation of 2,3,7,8-tetrachlor-odibenzo-p-dioxin hepatotoxicity with Ah phenotype in mice, Toxicol Appl Pharmacol, 74, pp. 17-25, (1984)

[5] Smith, Francis, Carthew, Iron as a synergist for hepatocellular carcinoma induced by polychlorinated biphenyls in Ah-responsive C57BL/10ScSn mice, Carcinogenesis, 11, pp. 437-444, (1990)

[6] Smith, Francis, Synergism of iron and hexachlorobenzene inhibits hepatic uroporphyrinogen decarboxylase in inbred mice with iron overload, Biochem J, 214, pp. 909-913, (1983)

[7] Rizzardini, Graziani, Carugo, Cantoni, Investigations on the role of free radical processes in hexachlorobenzene-induced porphyria in mice, J Biochem Toxicol, 3, pp. 33-46, (1988)

[8] Sweeney, Porphyria cutanea tarda, or the uroporphyrinogen decarboxylase deficiency diseases, Clin Biochem, 19, pp. 3-15, (1986)

[9] Francis, Smith, Polycyclic aromatic hydrocarbons cause hepatic porphyria in iron-loaded C57BL/ 10 mice: comparison of uroporphyrinogen decarboxylase inhibition with induction of alkoxyphenoxazone dealkylations, Biochem Biophys Res Commun, 146, pp. 13-20, (1987)

[10] Urquhart, Elder, Roberts, Lambrecht, Sinclair, Bement, Gorman, Sinclair, Uroporphyria produced in mice by 20-methylcholanthrene and 5-aminolevulinic acid, Biochem J, 253, pp. 357-362, (1988)