SLOW FORCE RECOVERY AFTER LONG-DURATION EXERCISE - METABOLIC AND ACTIVATION FACTORS IN MUSCLE FATIGUE

被引:124
作者
BAKER, AJ
KOSTOV, KG
MILLER, RG
WEINER, MW
机构
[1] CALIF PACIFIC MED CTR,DEPT NEUROL,SAN FRANCISCO,CA 94118
[2] UNIV CALIF SAN FRANCISCO,DEPT RADIOL,SAN FRANCISCO,CA 94121
关键词
CONTRACTION; P-31 NUCLEAR MAGNETIC RESONANCE; INORGANIC PHOSPHATE; EXCITATION-CONTRACTION COUPLING; ELECTROMYOGRAM;
D O I
10.1152/jappl.1993.74.5.2294
中图分类号
Q4 [生理学];
学科分类号
071003 ;
摘要
To investigate the roles of metabolic and nonmetabolic factors in human muscle fatigue, two relatively brief nonexhausting exercise protocols that produced similar levels of moderate fatigue were used: short-duration exercise (SDE; 2-min sustained maximal voluntary contraction) and long-duration exercise (LDE; 15- to 20-min intermittent exercise). After exercise and during recovery, multiple potential mechanisms of fatigue were studied from measurements of voluntary, twitch, and tetanic forces; intracellular metabolites (using P-31-nuclear magnetic resonance spectroscopy); and electromyographic signals. The major findings were as follows. 1) After SDE, fatigue closely correlated with increased [P(i)]. Both force and [P(i)] recovered within approximately 5 min after exercise. 2) After LDE, force recovered slowly, with significant fatigue beyond 15 min after exercise; however, recovery of [P(i)] was not slowed. 3) Electromyographic signals were little affected by either protocol. These findings suggest that multiple mechanisms contribute to moderate fatigue. Fatigue from SDE may arise primarily from metabolic mechanisms, whereas fatigue from LDE involves an additional slowly recovering nonmetabolic mechanism that may arise from impaired activation, beyond the cell membrane, at the level of excitation contraction coupling.
引用
收藏
页码:2294 / 2300
页数:7
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