REGULATION OF RAS SIGNAL-TRANSDUCTION IN NORMAL AND TRANSFORMED-CELLS

被引:30
作者
LOWE, PN
SKINNER, RH
机构
关键词
RAS; SIGNAL TRANSDUCTION; TRANSFORMATION; GTPASE-ACTIVATING PROTEIN; REGULATION; EXCHANGE FACTOR;
D O I
10.1016/0898-6568(94)90067-1
中图分类号
Q2 [细胞生物学];
学科分类号
071009 ; 090102 ;
摘要
引用
收藏
页码:109 / 123
页数:15
相关论文
共 143 条
[1]   DIFFERENTIAL REGULATION OF CELLULAR ACTIVITIES BY GTPASE-ACTIVATING PROTEIN AND NF1 [J].
ALALAWI, N ;
XU, GF ;
WHITE, R ;
CLARK, R ;
MCCORMICK, F ;
FERAMISCO, JR .
MOLECULAR AND CELLULAR BIOLOGY, 1993, 13 (04) :2497-2503
[2]   A CONSERVED ALTERNATIVE SPLICE IN THE VONRECKLINGHAUSEN NEUROFIBROMATOSIS (NF1) GENE PRODUCES 2 NEUROFIBROMIN ISOFORMS, BOTH OF WHICH HAVE GTPASE-ACTIVATING PROTEIN-ACTIVITY [J].
ANDERSEN, LB ;
BALLESTER, R ;
MARCHUK, DA ;
CHANG, E ;
GUTMANN, DH ;
SAULINO, AM ;
CAMONIS, J ;
WIGLER, M ;
COLLINS, FS .
MOLECULAR AND CELLULAR BIOLOGY, 1993, 13 (01) :487-495
[3]   MUTATIONS IN THE NEUROFIBROMATOSIS-1 GENE IN SPORADIC MALIGNANT-MELANOMA CELL-LINES [J].
ANDERSEN, LB ;
FOUNTAIN, JW ;
GUTMANN, DH ;
TARLE, SA ;
GLOVER, TW ;
DRACOPOLI, NC ;
HOUSMAN, DE ;
COLLINS, FS .
NATURE GENETICS, 1993, 3 (02) :118-121
[4]  
BARLAT I, 1993, ONCOGENE, V8, P215
[5]   MICROINJECTION OF THE RAS ONCOGENE PROTEIN INTO PC12 CELLS INDUCES MORPHOLOGICAL-DIFFERENTIATION [J].
BARSAGI, D ;
FERAMISCO, JR .
CELL, 1985, 42 (03) :841-848
[6]   ABERRANT REGULATION OF RAS PROTEINS IN MALIGNANT-TUMOR CELLS FROM TYPE-1 NEUROFIBROMATOSIS PATIENTS [J].
BASU, TN ;
GUTMANN, DH ;
FLETCHER, JA ;
GLOVER, TW ;
COLLINS, FS ;
DOWNWARD, J .
NATURE, 1992, 356 (6371) :713-715
[7]   CHARACTERIZATION OF FULL-LENGTH NEUROFIBROMIN - TUBULIN INHIBITS RAS GAP ACTIVITY [J].
BOLLAG, G ;
MCCORMICK, F ;
CLARK, R .
EMBO JOURNAL, 1993, 12 (05) :1923-1927
[8]   DIFFERENTIAL REGULATION OF RASGAP AND NEUROFIBROMATOSIS GENE-PRODUCT ACTIVITIES [J].
BOLLAG, G ;
MCCORMICK, F .
NATURE, 1991, 351 (6327) :576-579
[9]   THE SON OF SEVENLESS GENE-PRODUCT - A PUTATIVE ACTIVATOR OF RAS [J].
BONFINI, L ;
KARLOVICH, CA ;
DASGUPTA, C ;
BANERJEE, U .
SCIENCE, 1992, 255 (5044) :603-606
[10]  
BOS JL, 1989, CANCER RES, V49, P4682