MECHANISM AND PATHOGENESIS OF ISCHEMIA-INDUCED NEURONAL DAMAGE

[1] MECHANISM OF ARACHIDONIC-ACID LIBERATION DURING ISCHEMIA IN GERBIL CEREBRAL-CORTEX [J]. JOURNAL OF NEUROCHEMISTRY, 1987, 48 (02) : 503 - 509

[2] SELECTIVE INDUCTION OF KUNITZ-TYPE PROTEASE INHIBITOR DOMAIN-CONTAINING AMYLOID PRECURSOR PROTEIN MESSENGER-RNA AFTER PERSISTENT FOCAL ISCHEMIA IN RAT CEREBRAL-CORTEX [J]. NEUROSCIENCE LETTERS, 1991, 125 (02) : 172 - 174

[3] NUCLEOTIDE-SEQUENCE OF A BOVINE CLONE ENCODING THE ANGIOGENIC PROTEIN, BASIC FIBROBLAST GROWTH-FACTOR [J]. SCIENCE, 1986, 233 (4763) : 545 - 548

[4] TRANSLOCATION OF PROTEIN-KINASE-C ACTIVITY MAY MEDIATE HIPPOCAMPAL LONG-TERM POTENTIATION [J]. SCIENCE, 1986, 231 (4738) : 587 - 589

[5] N-METHYL-D-ASPARTATE ANTAGONISTS - READY FOR CLINICAL-TRIAL IN BRAIN ISCHEMIA [J]. ANNALS OF NEUROLOGY, 1989, 25 (04) : 398 - 403

[6] COMPARATIVE PROTECTIVE EFFECTS OF NICARDIPINE, FLUNARIZINE, LIDOFLAZINE AND NIMODIPINE AGAINST ISCHEMIC-INJURY IN THE HIPPOCAMPUS OF THE MONGOLIAN GERBIL [J]. BRITISH JOURNAL OF PHARMACOLOGY, 1988, 93 (04) : 877 - 883

[7] ALTURA BM, 1981, FED PROC, V40, P2672

[8] PATHO-PHYSIOLOGY OF ISCHEMIC CELL-DEATH .1. TIME OF ONSET OF IRREVERSIBLE DAMAGE - IMPORTANCE OF THE DIFFERENT COMPONENTS OF THE ISCHEMIC INSULT [J]. STROKE, 1983, 14 (02) : 219 - 226

[9] BASIC FIBROBLAST GROWTH-FACTOR PREVENTS DEATH OF LESIONED CHOLINERGIC NEURONS INVIVO [J]. NATURE, 1988, 332 (6162) : 360 - 361

[10] ENERGY-METABOLISM IN DELAYED NEURONAL DEATH OF CA1 NEURONS OF THE HIPPOCAMPUS FOLLOWING TRANSIENT ISCHEMIA IN THE GERBIL [J]. METABOLIC BRAIN DISEASE, 1986, 1 (04) : 263 - 278

[1] MECHANISM OF ARACHIDONIC-ACID LIBERATION DURING ISCHEMIA IN GERBIL CEREBRAL-CORTEX [J]. JOURNAL OF NEUROCHEMISTRY, 1987, 48 (02) : 503 - 509

[2] SELECTIVE INDUCTION OF KUNITZ-TYPE PROTEASE INHIBITOR DOMAIN-CONTAINING AMYLOID PRECURSOR PROTEIN MESSENGER-RNA AFTER PERSISTENT FOCAL ISCHEMIA IN RAT CEREBRAL-CORTEX [J]. NEUROSCIENCE LETTERS, 1991, 125 (02) : 172 - 174

[3] NUCLEOTIDE-SEQUENCE OF A BOVINE CLONE ENCODING THE ANGIOGENIC PROTEIN, BASIC FIBROBLAST GROWTH-FACTOR [J]. SCIENCE, 1986, 233 (4763) : 545 - 548

[4] TRANSLOCATION OF PROTEIN-KINASE-C ACTIVITY MAY MEDIATE HIPPOCAMPAL LONG-TERM POTENTIATION [J]. SCIENCE, 1986, 231 (4738) : 587 - 589

[5] N-METHYL-D-ASPARTATE ANTAGONISTS - READY FOR CLINICAL-TRIAL IN BRAIN ISCHEMIA [J]. ANNALS OF NEUROLOGY, 1989, 25 (04) : 398 - 403

[6] COMPARATIVE PROTECTIVE EFFECTS OF NICARDIPINE, FLUNARIZINE, LIDOFLAZINE AND NIMODIPINE AGAINST ISCHEMIC-INJURY IN THE HIPPOCAMPUS OF THE MONGOLIAN GERBIL [J]. BRITISH JOURNAL OF PHARMACOLOGY, 1988, 93 (04) : 877 - 883

[7] ALTURA BM, 1981, FED PROC, V40, P2672

[8] PATHO-PHYSIOLOGY OF ISCHEMIC CELL-DEATH .1. TIME OF ONSET OF IRREVERSIBLE DAMAGE - IMPORTANCE OF THE DIFFERENT COMPONENTS OF THE ISCHEMIC INSULT [J]. STROKE, 1983, 14 (02) : 219 - 226

[9] BASIC FIBROBLAST GROWTH-FACTOR PREVENTS DEATH OF LESIONED CHOLINERGIC NEURONS INVIVO [J]. NATURE, 1988, 332 (6162) : 360 - 361

[10] ENERGY-METABOLISM IN DELAYED NEURONAL DEATH OF CA1 NEURONS OF THE HIPPOCAMPUS FOLLOWING TRANSIENT ISCHEMIA IN THE GERBIL [J]. METABOLIC BRAIN DISEASE, 1986, 1 (04) : 263 - 278