HYPERSENSITIVITY AND REDUCED INHIBITION OF DNA-SYNTHESIS IN ATAXIA TELANGIECTASIA LYMPHOBLASTS TREATED WITH LOW-LEVELS OF NEOCARZINOSTATIN

被引:7
作者
BABILON, RW
SOPRANO, KJ
HENDERSON, EE
机构
来源
MUTATION RESEARCH | 1985年 / 146卷 / 01期
关键词
D O I
10.1016/0167-8817(85)90058-6
中图分类号
Q3 [遗传学];
学科分类号
071007 ; 090102 ;
摘要
引用
收藏
页码:79 / 87
页数:9
相关论文
共 42 条
[1]   RELATIONSHIP BETWEEN DNA STRAND-SCISSION AND DNA-SYNTHESIS INHIBITION IN HELA-CELLS TREATED WITH NEOCARZINOSTATIN [J].
BEERMAN, TA ;
GOLDBERG, IH .
BIOCHIMICA ET BIOPHYSICA ACTA, 1977, 475 (02) :281-293
[2]   SINGLE-STRAND NICKING OF DNA INVITRO BY NEOCARZINOSTATIN AND ITS POSSIBLE RELATIONSHIP TO MECHANISM OF DRUG ACTION [J].
BEERMAN, TA ;
POON, R ;
GOLDBERG, IH .
BIOCHIMICA ET BIOPHYSICA ACTA, 1977, 475 (02) :294-306
[3]   IDENTIFICATION OF ATAXIA TELANGIECTASIA HETEROZYGOTES, A CANCER PRONE POPULATION [J].
CHEN, PC ;
LAVIN, MF ;
KIDSON, C ;
MOSS, D .
NATURE, 1978, 274 (5670) :484-486
[4]   CYTOGENETIC INVESTIGATIONS IN FAMILIES WITH ATAXIA-TELANGIECTASIA [J].
COHEN, MM ;
SHAHAM, M ;
DAGAN, J ;
SHMUELI, E ;
KOHN, G .
CYTOGENETICS AND CELL GENETICS, 1975, 15 (05) :338-356
[5]   THE EFFECT OF BLEOMYCIN ON DNA-SYNTHESIS IN ATAXIA TELANGIECTASIA LYMPHOID-CELLS [J].
COHEN, MM ;
SIMPSON, SJ .
ENVIRONMENTAL MUTAGENESIS, 1982, 4 (01) :27-36
[6]   INCREASED CLASTOGENICITY AND DECREASED INHIBITION OF DNA-SYNTHESIS BY NEOCARZINOSTATIN AND TALLYSOMYCIN IN ATAXIA TELANGIECTASIA LYMPHOID-CELLS [J].
COHEN, MM ;
SIMPSON, SJ .
MUTATION RESEARCH, 1983, 112 (02) :119-128
[7]   BLEOMYCIN-RESISTANT DNA-SYNTHESIS IN ATAXIA TELANGIECTASIA CELLS [J].
CRAMER, P ;
PAINTER, RB .
NATURE, 1981, 291 (5817) :671-672
[8]   UNUSUAL LEVELS OF (ADP-RIBOSE)N AND DNA-SYNTHESIS IN ATAXIA TELANGIECTASIA CELLS FOLLOWING GAMMA-RAY IRRADIATION [J].
EDWARDS, MJ ;
TAYLOR, AMR .
NATURE, 1980, 287 (5784) :745-747
[9]  
GOLDBERG IH, 1981, MOL ACTIONS TARGETS, P163
[10]   MODE OF INHIBITION OF DNA-REPLICATION IN NEOCARZINOSTATIN-TREATED HELA-CELLS [J].
HATAYAMA, T ;
YUKIOKA, M .
BIOCHIMICA ET BIOPHYSICA ACTA, 1983, 740 (03) :291-299