Endoplasmic Reticulum Stress-Related Inflammation and Cardiovascular Diseases

被引:101
作者
Gotoh, Tomomi [1 ,2 ]
Endo, Motoyoshi [1 ]
Oike, Yuichi [1 ]
机构
[1] Kumamoto Univ, Grad Sch Med Sci, Dept Mol Genet, Honjo 1-1-1, Kumamoto 8608556, Japan
[2] Kumamoto Univ, Dept Sch Hlth, Fac Educ, Kumamoto 8608555, Japan
关键词
D O I
10.4061/2011/259462
中图分类号
R392 [医学免疫学]; Q939.91 [免疫学];
学科分类号
100102 [免疫学];
摘要
The endoplasmic reticulum (ER) is the site of synthesis and maturation of proteins designed for secretion or for localization on the cell membrane. Various types of stress from both inside and outside cells disturb ER function, thus causing unfolded or misfolded proteins to accumulate in the ER. To improve and maintain the ER functions against such stresses, the ER stress response pathway is activated. However, when the stress is prolonged or severe, apoptosis pathways are activated to remove damaged cells. It was recently reported that the ER stress pathway is also involved in the inflammatory response, whereby inflammation induces ER stress, and ER stress induces an inflammatory response. Therefore, the ER stress response pathway is involved in various diseases, including cardiovascular diseases such as atherosclerosis and ischemic diseases, in various ways. The ER stress pathway may represent a novel target for the treatment of these diseases.
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页数:8
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