THE EFFECT OF INDUCERS OF MIXED-FUNCTION OXIDASES ON HEPATIC MICROSOME-MEDIATED AFLATOXIN-B1 TRANSFORMATION IN C3H-10T1/2 CELLS

被引：10

作者：

FALETTO, MB ^{[1
]}

GURTOO, HL ^{[1
]}

机构：

[1] NEW YORK STATE DEPT HLTH,ROSWELL PK MEM INST,GRACE CANC DRUG CTR,ELM & CARLTON ST,BUFFALO,NY 14263

来源：

TOXICOLOGY AND APPLIED PHARMACOLOGY | 1989年 / 98卷 / 02期

关键词：

D O I：

10.1016/0041-008X(89)90230-5

中图分类号：

R9 [药学];

学科分类号：

1007 ;

摘要：

引用

页码：252 / 262

页数：11

共 45 条

[1]

AMSTAD PA, 1983, J NATL CANCER I, V70, P135

[2] S-9 METABOLIC-ACTIVATION ENHANCES AFLATOXIN-MEDIATED TRANSFORMATION OF C3H-10T1/2 CELLS [J].

BILLINGS, PC ;

HEIDELBERGER, C ;

LANDOLPH, JR .

TOXICOLOGY AND APPLIED PHARMACOLOGY, 1985, 77 (01) :58-65

[3]

BRADFORD MM, 1976, ANAL BIOCHEM, V72, P248, DOI 10.1016/0003-2697(76)90527-3

[4]

Busby WF, 1984, ACS MONOGR SER, V182, P945

[5] COMPARATIVE MUTAGENICITY OF AFLATOXINS USING A SALMONELLA TROUT HEPATIC ENZYME ACTIVATION SYSTEM [J].

COULOMBE, RA ;

SHELTON, DW ;

SINNHUBER, RO ;

NIXON, JE .

CARCINOGENESIS, 1982, 3 (11) :1261-1264

[6]

CROY RG, 1981, CANCER RES, V41, P197

[7]

CULLEN JM, 1987, CANCER RES, V47, P1913

[8] DIFFERENCES IN AFLATOXIN-B1-SUSCEPTIBILITY OF RAT AND MOUSE ARE CORRELATED WITH THE CAPABILITY INVITRO TO INACTIVATE AFLATOXIN-B1-EPOXIDE [J].

DEGEN, GH ;

NEUMANN, HG .

CARCINOGENESIS, 1981, 2 (04) :299-306

[9] MAJOR METABOLITE OF AFLATOXIN-B1 IN RAT IS A GLUTATHIONE CONJUGATE [J].

DEGEN, GH ;

NEUMANN, HG .

CHEMICO-BIOLOGICAL INTERACTIONS, 1978, 22 (2-3) :239-255

[10]

DENT JG, 1979, BANBURY REPORT, V2, P295

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