THE INVOLVEMENT OF ONCOGENES AND TUMOR SUPPRESSOR GENES IN THE CONTROL OF APOPTOSIS

被引:107
作者
OREN, M
机构
[1] Department of Chemical Immunology, The Weizmann Institute of Science, Rehovot
关键词
APOPTOSIS; BCL-2; ONCOGENES; P53; TUMOR SUPPRESSOR GENES;
D O I
10.1007/BF00048060
中图分类号
R73 [肿瘤学];
学科分类号
100214 ;
摘要
[No abstract available]
引用
收藏
页码:141 / 148
页数:8
相关论文
共 46 条
[1]  
ARENDS MJ, 1991, INT REV EXP PATHOL, V32, P223
[2]  
ASKEW DS, 1991, ONCOGENE, V6, P1915
[3]   SUPPRESSION OF HUMAN COLORECTAL-CARCINOMA CELL-GROWTH BY WILD-TYPE-P53 [J].
BAKER, SJ ;
MARKOWITZ, S ;
FEARON, ER ;
WILLSON, JKV ;
VOGELSTEIN, B .
SCIENCE, 1990, 249 (4971) :912-915
[4]   DETERMINATION OF THE LENGTH OF THE HISTOLOGICAL STAGES OF APOPTOSIS IN NORMAL LIVER AND IN ALTERED HEPATIC FOCI OF RATS [J].
BURSCH, W ;
PAFFE, S ;
PUTZ, B ;
BARTHEL, G ;
SCHULTEHERMANN, R .
CARCINOGENESIS, 1990, 11 (05) :847-853
[5]  
COLLINS M, 1991, Current Biology, V1, P140, DOI 10.1016/0960-9822(91)90212-F
[6]   P53 FUNCTIONS AS A CELL-CYCLE CONTROL PROTEIN IN OSTEOSARCOMAS [J].
DILLER, L ;
KASSEL, J ;
NELSON, CE ;
GRYKA, MA ;
LITWAK, G ;
GEBHARDT, M ;
BRESSAC, B ;
OZTURK, M ;
BAKER, SJ ;
VOGELSTEIN, B ;
FRIEND, SH .
MOLECULAR AND CELLULAR BIOLOGY, 1990, 10 (11) :5772-5781
[7]   WILD-TYPE P53 CAN INHIBIT ONCOGENE-MEDIATED FOCUS FORMATION [J].
ELIYAHU, D ;
MICHALOVITZ, D ;
ELIYAHU, S ;
PINHASIKIMHI, O ;
OREN, M .
PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA, 1989, 86 (22) :8763-8767
[8]   MECHANISMS AND FUNCTIONS OF CELL-DEATH [J].
ELLIS, RE ;
YUAN, JY ;
HORVITZ, HR .
ANNUAL REVIEW OF CELL BIOLOGY, 1991, 7 :663-698
[9]   THE P53 PROTO-ONCOGENE CAN ACT AS A SUPPRESSOR OF TRANSFORMATION [J].
FINLAY, CA ;
HINDS, PW ;
LEVINE, AJ .
CELL, 1989, 57 (07) :1083-1093
[10]   CELL-DEATH MECHANISMS AND THE IMMUNE-SYSTEM [J].
GOLSTEIN, P ;
OJCIUS, DM ;
YOUNG, JDE .
IMMUNOLOGICAL REVIEWS, 1991, 121 :29-65