DECREASED HOST-CELL REACTIVATION OF UV-IRRADIATED ADENOVIRUS-5 BY FIBROBLASTS FROM COCKAYNES SYNDROME PATIENTS

被引：16

作者：

DAY, RS

ZIOLKOWSKI, CHJ

DIMATTINA, M

机构：

来源：

PHOTOCHEMISTRY AND PHOTOBIOLOGY | 1981年 / 34卷 / 05期

关键词：

D O I：

10.1111/j.1751-1097.1981.tb09049.x

中图分类号：

Q5 [生物化学]; Q7 [分子生物学];

学科分类号：

071010 ; 081704 ;

摘要：

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页码：603 / 607

页数：5

相关论文

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[1] COCKAYNE SYNDROME FIBROBLASTS HAVE INCREASED SENSITIVITY TO ULTRAVIOLET-LIGHT BUT NORMAL RATES OF UNSCHEDULED DNA-SYNTHESIS
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[3] COKER S, 1979, NEUROOPHTHALMOLOGY F, P379
[4] HUMAN-TUMOR CELL STRAINS DEFECTIVE IN THE REPAIR OF ALKYLATION DAMAGE
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[5] HUMAN-BRAIN TUMOR-CELL STRAINS WITH DEFICIENT HOST-CELL REACTIVATION OF N-METHYL-N'-NITRO-N-NITROSOGUANIDINE-DAMAGED ADENOVIRUS-5
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[J]. NATURE, 1979, 279 (5716) : 797 - 799
[6] DEFECTIVE REPAIR OF ALKYLATED DNA BY HUMAN-TUMOR AND SV40-TRANSFORMED HUMAN CELL STRAINS
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[7] DAY RS, 1974, CANCER RES, V34, P1965
[8] EXCISION REPAIR BY HUMAN FIBROBLASTS OF DNA DAMAGED BY R-7,T-8-DIHYDROXY -T-9,10-OXY-7,8,9,10-TETRAHYDROBENZO(A)PYRENE
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[J]. MUTATION RESEARCH, 1978, 50 (03): : 383 - 394
[9] DAY RS, 1978, DNA REPAIR MECHANISM, P535
[10] HOST-CELL REACTIVATION OF UV-IRRADIATED ADENOVIRUS IN COCKAYNE SYNDROME
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