THE CELLULAR BASIS FOR LIVER-INJURY IN ALPHA-1-ANTITRYPSIN DEFICIENCY

[1] STUDIES OF HEPATIC SYNTHESIS INVIVO OF PLASMA-PROTEINS, INCLUDING OROSOMUCOID, TRANSFERRIN, ALPHA-1-ANTITRYPSIN, C8, AND FACTOR-B [J]. CLINICAL IMMUNOLOGY AND IMMUNOPATHOLOGY, 1980, 16 (01): : 84 - 89

[2] ABNORMAL PROTEINS SERVE AS EUKARYOTIC STRESS SIGNALS AND TRIGGER THE ACTIVATION OF HEAT-SHOCK GENES [J]. SCIENCE, 1986, 232 (4749) : 522 - 524

[3] ONCOGENE JUN ENCODES A SEQUENCE-SPECIFIC TRANS-ACTIVATOR SIMILAR TO AP-1 [J]. NATURE, 1988, 332 (6160) : 166 - 171

[4] SEQUENTIAL CHANGES OF PLASMA-PROTEINS AFTER SURGICAL TRAUMA [J]. SCANDINAVIAN JOURNAL OF CLINICAL & LABORATORY INVESTIGATION, 1972, 29 : 127 - &

[5] REGULATION OF ALPHA-1 PROTEINASE-INHIBITOR FUNCTION BY RABBIT ALVEOLAR MACROPHAGES - EVIDENCE FOR PROTEOLYTIC RATHER THAN OXIDATIVE INACTIVATION [J]. JOURNAL OF CLINICAL INVESTIGATION, 1985, 75 (06) : 1758 - 1762

[6] THE INHIBITORY COMPLEX OF HUMAN ALPHA-1-PROTEINASE INHIBITOR AND HUMAN-LEUKOCYTE ELASTASE IS A NEUTROPHIL CHEMOATTRACTANT [J]. JOURNAL OF EXPERIMENTAL MEDICINE, 1988, 167 (05) : 1608 - 1615

[7] BANDA MJ, 1988, J BIOL CHEM, V263, P4481

[8] LIMITED PROTEOLYSIS BY MACROPHAGE ELASTASE INACTIVATES HUMAN ALPHA-1-PROTEINASE INHIBITOR [J]. JOURNAL OF EXPERIMENTAL MEDICINE, 1980, 152 (06) : 1563 - 1570

[9] LIPOPOLYSACCHARIDE MODULATES THE EXPRESSION OF ALPHA-1 PROTEINASE-INHIBITOR AND OTHER SERINE PROTEINASE-INHIBITORS IN HUMAN-MONOCYTES AND MACROPHAGES [J]. JOURNAL OF EXPERIMENTAL MEDICINE, 1987, 166 (04) : 1041 - 1054

[10] HUMAN Z-ALPHA-1-ANTITRYPSIN ACCUMULATES INTRACELLULARLY AND STIMULATES LYSOSOMAL ACTIVITY WHEN SYNTHESIZED IN THE XENOPUS OOCYTE [J]. FEBS LETTERS, 1985, 183 (02) : 304 - 308

[1] STUDIES OF HEPATIC SYNTHESIS INVIVO OF PLASMA-PROTEINS, INCLUDING OROSOMUCOID, TRANSFERRIN, ALPHA-1-ANTITRYPSIN, C8, AND FACTOR-B [J]. CLINICAL IMMUNOLOGY AND IMMUNOPATHOLOGY, 1980, 16 (01): : 84 - 89

[2] ABNORMAL PROTEINS SERVE AS EUKARYOTIC STRESS SIGNALS AND TRIGGER THE ACTIVATION OF HEAT-SHOCK GENES [J]. SCIENCE, 1986, 232 (4749) : 522 - 524

[3] ONCOGENE JUN ENCODES A SEQUENCE-SPECIFIC TRANS-ACTIVATOR SIMILAR TO AP-1 [J]. NATURE, 1988, 332 (6160) : 166 - 171

[4] SEQUENTIAL CHANGES OF PLASMA-PROTEINS AFTER SURGICAL TRAUMA [J]. SCANDINAVIAN JOURNAL OF CLINICAL & LABORATORY INVESTIGATION, 1972, 29 : 127 - &

[5] REGULATION OF ALPHA-1 PROTEINASE-INHIBITOR FUNCTION BY RABBIT ALVEOLAR MACROPHAGES - EVIDENCE FOR PROTEOLYTIC RATHER THAN OXIDATIVE INACTIVATION [J]. JOURNAL OF CLINICAL INVESTIGATION, 1985, 75 (06) : 1758 - 1762

[6] THE INHIBITORY COMPLEX OF HUMAN ALPHA-1-PROTEINASE INHIBITOR AND HUMAN-LEUKOCYTE ELASTASE IS A NEUTROPHIL CHEMOATTRACTANT [J]. JOURNAL OF EXPERIMENTAL MEDICINE, 1988, 167 (05) : 1608 - 1615

[7] BANDA MJ, 1988, J BIOL CHEM, V263, P4481

[8] LIMITED PROTEOLYSIS BY MACROPHAGE ELASTASE INACTIVATES HUMAN ALPHA-1-PROTEINASE INHIBITOR [J]. JOURNAL OF EXPERIMENTAL MEDICINE, 1980, 152 (06) : 1563 - 1570

[9] LIPOPOLYSACCHARIDE MODULATES THE EXPRESSION OF ALPHA-1 PROTEINASE-INHIBITOR AND OTHER SERINE PROTEINASE-INHIBITORS IN HUMAN-MONOCYTES AND MACROPHAGES [J]. JOURNAL OF EXPERIMENTAL MEDICINE, 1987, 166 (04) : 1041 - 1054

[10] HUMAN Z-ALPHA-1-ANTITRYPSIN ACCUMULATES INTRACELLULARLY AND STIMULATES LYSOSOMAL ACTIVITY WHEN SYNTHESIZED IN THE XENOPUS OOCYTE [J]. FEBS LETTERS, 1985, 183 (02) : 304 - 308