REGULATION OF EUKARYOTIC GENE-EXPRESSION BY TRANSCRIPTIONAL ATTENUATION

被引：40

作者：

WRIGHT, S

机构：

[1] CRC Cancer/Devtl. Biology Institute

来源：

MOLECULAR BIOLOGY OF THE CELL | 1993年 / 4卷 / 07期

关键词：

D O I：

10.1091/mbc.4.7.661

中图分类号：

Q2 [细胞生物学];

学科分类号：

071009 ; 090102 ;

摘要：

[No abstract available]

引用

页码：661 / 668

页数：8

共 114 条

[1]

ALIFANO P, 1991, CELL, V64, P553

[2] ATTENUATION MAY REGULATE GENE-EXPRESSION IN ANIMAL VIRUSES AND CELLS [J].

ALONI, Y ;

HAY, N .

CRC CRITICAL REVIEWS IN BIOCHEMISTRY, 1985, 18 (04) :327-383

[3] MODULATION OF THE DIMERIZATION OF A TRANSCRIPTIONAL ANTITERMINATOR PROTEIN BY PHOSPHORYLATION [J].

AMSTERCHODER, O ;

WRIGHT, A .

SCIENCE, 1992, 257 (5075) :1395-1398

[4] TRANSCRIPTIONAL TERMINATION BETWEEN THE CLOSELY LINKED HUMAN-COMPLEMENT GENES C2 AND FACTOR-B - COMMON TERMINATION FACTOR FOR C2 AND C-MYC [J].

ASHFIELD, R ;

ENRIQUEZHARRIS, P ;

PROUDFOOT, NJ .

EMBO JOURNAL, 1991, 10 (13) :4197-4207

[5] AN ANTITERMINATION PROTEIN ENGAGES THE ELONGATING TRANSCRIPTION APPARATUS AT A PROMOTER PROXIMAL RECOGNITION SITE [J].

BARIK, S ;

GHOSH, B ;

WHALEN, W ;

LAZINSKI, D ;

DAS, A .

CELL, 1987, 50 (06) :885-899

[6] DIFFERENTIAL EXPRESSION OF C-MYB MESSENGER-RNA IN MURINE-B LYMPHOMAS BY A BLOCK TO TRANSCRIPTION ELONGATION [J].

BENDER, TP ;

THOMPSON, CB ;

KUEHL, WM .

SCIENCE, 1987, 237 (4821) :1473-1476

[7] ROLE OF THE MAMMALIAN TRANSCRIPTION FACTOR-IIF, FACTOR-IIS, AND FACTOR-IIX DURING ELONGATION BY RNA POLYMERASE-II [J].

BENGAL, E ;

FLORES, O ;

KRAUSKOPF, A ;

REINBERG, D ;

ALONI, Y .

MOLECULAR AND CELLULAR BIOLOGY, 1991, 11 (03) :1195-1206

[8] TRANSCRIPTIONAL ELONGATION BY PURIFIED RNA POLYMERASE-II IS BLOCKED AT THE TRANS-ACTIVATION-RESPONSIVE REGION OF HUMAN-IMMUNODEFICIENCY-VIRUS TYPE-1 INVITRO [J].

BENGAL, E ;

ALONI, Y .

JOURNAL OF VIROLOGY, 1991, 65 (09) :4910-4918

[9] SEQUENCE REQUIREMENTS FOR PREMATURE TERMINATION OF TRANSCRIPTION IN THE HUMAN C-MYC GENE [J].

BENTLEY, DL ;

GROUDINE, M .

CELL, 1988, 53 (02) :245-256

[10] A BLOCK TO ELONGATION IS LARGELY RESPONSIBLE FOR DECREASED TRANSCRIPTION OF C-MYC IN DIFFERENTIATED HL60 CELLS [J].

BENTLEY, DL ;

GROUDINE, M .

NATURE, 1986, 321 (6071) :702-706

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