ACRYLAMIDE TREATMENT OF PTKL CELLS CAUSES DEPHOSPHORYLATION OF KERATIN POLYPEPTIDES

被引：25

作者：

ECKERT, BS ^{[1
]}

YEAGLE, PL ^{[1
]}

机构：

[1] SUNY BUFFALO,SCH MED,DEPT BIOCHEM,BUFFALO,NY 14214

来源：

CELL MOTILITY AND THE CYTOSKELETON | 1988年 / 11卷 / 01期

关键词：

D O I：

10.1002/cm.970110104

中图分类号：

Q2 [细胞生物学];

学科分类号：

071009 ; 090102 ;

摘要：

引用

页码：24 / 30

页数：7

共 24 条

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[2]

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[3] ALTERATION OF THE DISTRIBUTION OF INTERMEDIATE FILAMENTS IN PTK1 CELLS BY ACRYLAMIDE .2. EFFECT ON THE ORGANIZATION OF CYTOPLASMIC ORGANELLES [J].

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[4]

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[7] DIFFERENTIAL POST-TRANSLATIONAL MODIFICATION OF HUMAN TYPE-I KERATINS SYNTHESIZED IN A RABBIT RETICULOCYTE CELL-FREE SYSTEM [J].

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[8] PHOSPHORYLATION OF EPIDERMAL KERATINS [J].

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[9] DUAL REGULATION OF INTERMEDIATE FILAMENT PHOSPHORYLATION [J].

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[10] ALTERED PHOSPHORYLATION OF RAT NEURONAL CYTOSKELETAL PROTEINS IN ACRYLAMIDE INDUCED NEUROPATHY [J].

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ALLI, P .

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