RIGHT ATRIAL AND VENTRICULAR INFARCTION - EVALUATION WITH TRANSESOPHAGEAL ECHOCARDIOGRAPHY

被引：9

作者：

GOMEZVILLALOBOS, MD ^{[1
]}

VARGASBARRON, J ^{[1
]}

ROMEROCARDENAS, A ^{[1
]}

RIJLAARSDAM, M ^{[1
]}

KEIRNS, C ^{[1
]}

LUPIHERRERA, E ^{[1
]}

GONZALEZESPINO, J ^{[1
]}

机构：

[1] INST NACL CARDIOL IGNACIO CHAVEZ,DEPT ECHOCARDIOG,MEXICO CITY 14080,DF,MEXICO

来源：

ECHOCARDIOGRAPHY-A JOURNAL OF CARDIOVASCULAR ULTRASOUND AND ALLIED TECHNIQUES | 1995年 / 12卷 / 02期

关键词：

RIGHT ATRIAL INFARCTION; RIGHT VENTRICULAR INFARCTION; TRANSESOPHAGEAL ECHOCARDIOGRAPHY;

D O I：

10.1111/j.1540-8175.1995.tb00531.x

中图分类号：

R5 [内科学];

学科分类号：

1002 ; 100201 ;

摘要：

In order to compare the results of transesophageal echocardiography (TEE) in diagnosis of right atrial (RA) and right ventricular (RV) infarction with those of transthoracic echocardiography (TTE), 11 patients admitted to the coronary care unit with the diagnosis of posteroinferior left ventricular (LV) acute myocardial infarction (MI) and electrocardiographic suspicion of extension, to RV were studied. In two of the 11 patients, RA infarction was identified on the basis of akinesis of the RA free wall, dilatation of the atrial cavity, spontaneous echo contrast, mural thrombosis, and poor atrial contribution to RV filling. In all 11 patients, RV infarction was determined by akinesis of one or more segmental regions, dilatation. of the cavity in four patients, and tricuspid regurgitation in vides additional information to TTE for determining RA and RV infarction during the early stages of MI.

引用

页码：129 / 137

页数：9

共 19 条

[1]

Isner J, Roberts W, Right ventricular infarction complicating left ventricular infarction secondary to coronary heart disease, Am J Cardiol, 42, pp. 885-894, (1978)

[2]

Isner J, Roberts W, Right ventricular infarction complicating left ventricular infarction: A complication exclusively of posterior wall infarction, The American Journal of Cardiology, 41, pp. 409-415, (1978)

[3]

Ratliff NB, Hackel DB, Combined right and left ventricular infarction: Pathogenesis and clinicopathologic correlations, Am J Cardiol, 45, pp. 218-221, (1980)

[4]

Gardin JM, Singer DH, Atrial infarction. Importance, diagnosis, and localization, Arch Intern Med, 141, pp. 1345-1348, (1981)

[5]

Lazar EJ, Goldberg J, Peled H, Atrial infarction: Diagnosis and management, Am Heart J, 116, pp. 1058-1063, (1988)

[6]

Cardenas M, del Diaz, Gonzalez, Et al., El infarto agudo del miocardio del ventriculo derecho, Arch Inst Cardiol Mex, 50, pp. 295-321, (1980)

[7]

Medrano G, De Micheli A, Osornio A, Repercusiones del infarto experimental de la AD sobre P, Ta y Ota, Arch Inst Cardiol Mex, 55, pp. 297-303, (1985)

[8]

Medrano G, De Micheli A, Osornio A, Interatrial conduction STa in experimental atrial damage, J Electrocardiol, 20, pp. 357-362, (1987)

[9]

Cohn JN, Guija NH, Broder MI, Et al., Right ventricular infarction. Clinical and hemodynamic features, Am J Cardiol, 33, pp. 209-214, (1974)

[10]

Baigrie R, Morgan H, Et al., The spectrum of right ventricular movement in inferior wall myocardial infarction: A clinical, hemodynamic and noninvasive study, JACC, 1, pp. 1396-1404, (1983)

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