SIGNAL-TRANSDUCTION - THE HUNT FOR RAS TARGETS

被引：65

作者：

FEIG, LA ^{[1
]}

SCHAFFHAUSEN, B ^{[1
]}

机构：

[1] TUFTS UNIV,SCH MED,SACKLER SCH GRAD BIOMED SCI,BOSTON,MA 02111

来源：

NATURE | 1994年 / 370卷 / 6490期

关键词：

D O I：

10.1038/370508a0

中图分类号：

O [数理科学和化学]; P [天文学、地球科学]; Q [生物科学]; N [自然科学总论];

学科分类号：

07 ; 0710 ; 09 ;

摘要：

引用

页码：508 / 509

页数：2

共 21 条

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[5] DISRUPTION OF PDGF RECEPTOR TRAFFICKING BY MUTATION OF ITS PI-3 KINASE BINDING-SITES
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[J]. SCIENCE, 1994, 263 (5147) : 684 - 687
[6] KUMAGAI N, 1993, J BIOL CHEM, V268, P24535
[7] REQUIREMENT FOR RAS IN RAF ACTIVATION IS OVERCOME BY TARGETING RAF TO THE PLASMA-MEMBRANE
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[J]. NATURE, 1994, 369 (6479) : 411 - 414
[8] TRANSFORMATION-DEFECTIVE MUTANTS OF POLYOMAVIRUS MIDDLE T-ANTIGEN ASSOCIATE WITH PHOSPHATIDYLINOSITOL 3-KINASE (PI 3-KINASE) BUT ARE UNABLE TO MAINTAIN WILD-TYPE LEVELS OF PI 3-KINASE PRODUCTS IN INTACT-CELLS
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[9] LIPID 2ND MESSENGERS
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[J]. CELL, 1994, 77 (03) : 329 - 334
[10] FUNCTION AND REGULATION OF RAS
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[J]. ANNUAL REVIEW OF BIOCHEMISTRY, 1993, 62 : 851 - 891

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