REGULATION OF BIOSYNTHESIS AND PHOSPHORYLATION OF P210BCR ABL PROTEIN DURING DIFFERENTIATION INDUCTION OF K-562 CELLS

被引：21

作者：

NISHIMURA, J ^{[1
]}

TAKAHIRA, H ^{[1
]}

SHIBATA, K ^{[1
]}

MUTA, K ^{[1
]}

YAMAMOTO, M ^{[1
]}

IDEGUCHI, H ^{[1
]}

UMEMURA, T ^{[1
]}

NAWATA, H ^{[1
]}

机构：

[1] KYUSHU UNIV,SCH HLTH SCI,DEPT MED TECHNOL,FUKUOKA 812,JAPAN

来源：

LEUKEMIA RESEARCH | 1988年 / 12卷 / 11-12期

关键词：

D O I：

10.1016/0145-2126(88)90014-8

中图分类号：

R73 [肿瘤学];

学科分类号：

100214 ;

摘要：

引用

页码：875 / &

共 26 条

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[7]

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[8] THE CML-SPECIFIC P210 BCR/ABL PROTEIN, UNLIKE V-ABL, DOES NOT TRANSFORM NIH/3T3 FIBROBLASTS [J].

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[9]

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[10] CELL-LINES AND CLINICAL ISOLATES DERIVED FROM PH1-POSITIVE CHRONIC MYELOGENOUS LEUKEMIA PATIENTS EXPRESS C-ABL PROTEINS WITH A COMMON STRUCTURAL ALTERATION [J].

KONOPKA, JB ;

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