Calcium transport in the kidney

被引:20
作者
Friedman, PA
机构
[1] Univ Pittsburgh, Sch Med, Dept Pharmacol, Pittsburgh, PA 15261 USA
[2] Univ Pittsburgh, Sch Med, Dept Med, Pittsburgh, PA 15261 USA
关键词
D O I
10.1097/00041552-199909000-00010
中图分类号
R5 [内科学]; R69 [泌尿科学(泌尿生殖系疾病)];
学科分类号
1002 ; 100201 ;
摘要
Recent discoveries of calcium-regulating and calcium-transporting proteins have paved the way for a heightened understanding of the mechanisms and control of renal calcium transport. In this review, new findings regarding the multifunctional megalin receptor, chloride channels, a putative calcium entry channel, and the calcium-sensing receptor are discussed. Curr Opin Nephrol Hypertens 8:589-595. (C) 1999 Lippincott Williams & Wilkins.
引用
收藏
页码:589 / 595
页数:7
相关论文
共 58 条
[31]   VITAMIN-D PLASMA-BINDING PROTEIN - TURNOVER AND FATE IN THE RABBIT [J].
HADDAD, JG ;
FRASER, DR ;
LAWSON, DEM .
JOURNAL OF CLINICAL INVESTIGATION, 1981, 67 (05) :1550-1560
[32]   PLASMA VITAMIN-D-BINDING PROTEIN (GC-GLOBULIN) - MULTIPLE TASKS [J].
HADDAD, JG .
JOURNAL OF STEROID BIOCHEMISTRY AND MOLECULAR BIOLOGY, 1995, 53 (1-6) :579-582
[33]   Urinary calcium excretion and renal calbindin-D28k [J].
Hemmingsen, C ;
Staun, M ;
Meibom, K ;
Bang, K ;
Olgaard, K .
KIDNEY & BLOOD PRESSURE RESEARCH, 1997, 20 (04) :247-250
[34]   Megalin antagonizes activation of the parathyroid hormone receptor [J].
Hilpert, J ;
Nykjaer, A ;
Jacobsen, C ;
Wallukat, G ;
Nielsen, R ;
Moestrup, SK ;
Haller, H ;
Luft, FC ;
Christensen, EI ;
Willnow, TE .
JOURNAL OF BIOLOGICAL CHEMISTRY, 1999, 274 (09) :5620-5625
[35]   Molecular identification of the apical Ca2+ channel in 1,25-dihydroxyvitamin D3-responsive epithelia [J].
Hoenderop, JGJ ;
van der Kemp, AWCM ;
Hartog, A ;
van de Graaf, SFJ ;
van Os, CH ;
Willems, PHGM ;
Bindels, RJM .
JOURNAL OF BIOLOGICAL CHEMISTRY, 1999, 274 (13) :8375-8378
[36]   TRANSPORT AND CATABOLISM OF PARATHYROID-HORMONE IN ISOLATED RAT-KIDNEY [J].
KAU, ST ;
MAACK, T .
AMERICAN JOURNAL OF PHYSIOLOGY, 1977, 233 (05) :F445-F454
[37]   2 HIGHLY HOMOLOGOUS MEMBERS OF THE CLC CHLORIDE CHANNEL FAMILY IN BOTH RAT AND HUMAN KIDNEY [J].
KIEFERLE, S ;
FONG, PY ;
BENS, M ;
VANDEWALLE, A ;
JENTSCH, TJ .
PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA, 1994, 91 (15) :6943-6947
[38]   Idiopathic low molecular weight proteinuria associated with hypercalciuric nephrocalcinosis in Japanese children is due to mutations of the renal chloride channel (CLCN5) [J].
Lloyd, SE ;
Pearce, SHS ;
Gunther, W ;
Kawaguchi, H ;
Igarashi, T ;
Jentsch, TJ ;
Thakker, RV .
JOURNAL OF CLINICAL INVESTIGATION, 1997, 99 (05) :967-974
[39]   Characterisation of renal chloride channel, CLCN5, mutations in hypercalciuric nephrolithiasis (kidney stones) disorders [J].
Lloyd, SE ;
Gunther, W ;
Pearce, SHS ;
Thomson, A ;
Bianchi, ML ;
Bosio, M ;
Craig, IW ;
Fisher, SE ;
Scheinman, SJ ;
Wrong, O ;
Jentsch, TJ ;
Thakker, RV .
HUMAN MOLECULAR GENETICS, 1997, 6 (08) :1233-1239
[40]   A common molecular basis for three inherited kidney stone diseases [J].
Lloyd, SE ;
Pearce, SHS ;
Fisher, SE ;
Steinmeyer, K ;
Schwappach, B ;
Scheinman, SJ ;
Harding, B ;
Bolino, A ;
Devoto, M ;
Goodyer, P ;
Rigden, SPA ;
Wrong, O ;
Jentsch, TJ ;
Craig, IW ;
Thakker, RV .
NATURE, 1996, 379 (6564) :445-449