CELL-CYCLE - DAMS AND SLUICES

[1] CHENG HC, 1986, J BIOL CHEM, V261, P989

[2] WAF1, A POTENTIAL MEDIATOR OF P53 TUMOR SUPPRESSION [J]. CELL, 1993, 75 (04) : 817 - 825

[3] MUTATION OF FISSION YEAST-CELL CYCLE CONTROL GENES ABOLISHES DEPENDENCE OF MITOSIS ON DNA-REPLICATION [J]. CELL, 1990, 60 (04) : 665 - 673

[4] INHIBITION OF CDK2 ACTIVITY IN-VIVO BY AN ASSOCIATED 20K REGULATORY SUBUNIT [J]. NATURE, 1993, 366 (6456) : 707 - 710

[5] HARPER JW, 1993, CELL, V75, P805

[6] DEFECTS IN A CELL-CYCLE CHECKPOINT MAY BE RESPONSIBLE FOR THE GENOMIC INSTABILITY OF CANCER-CELLS [J]. CELL, 1992, 71 (04) : 543 - 546

[7] HARTWELL LH, 1991, GENETICS, V129, P975

[8] JENTSCH D, 1992, A REV GENET, V26, P179

[9] A MAMMALIAN-CELL CYCLE CHECKPOINT PATHWAY UTILIZING P53 AND GADD45 IS DEFECTIVE IN ATAXIA-TELANGIECTASIA [J]. CELL, 1992, 71 (04) : 587 - 597

[10] CHECKPOINTS THAT COUPLE GENE-EXPRESSION TO MORPHOGENESIS [J]. SCIENCE, 1993, 262 (5137) : 1227 - 1228

[1] CHENG HC, 1986, J BIOL CHEM, V261, P989

[2] WAF1, A POTENTIAL MEDIATOR OF P53 TUMOR SUPPRESSION [J]. CELL, 1993, 75 (04) : 817 - 825

[3] MUTATION OF FISSION YEAST-CELL CYCLE CONTROL GENES ABOLISHES DEPENDENCE OF MITOSIS ON DNA-REPLICATION [J]. CELL, 1990, 60 (04) : 665 - 673

[4] INHIBITION OF CDK2 ACTIVITY IN-VIVO BY AN ASSOCIATED 20K REGULATORY SUBUNIT [J]. NATURE, 1993, 366 (6456) : 707 - 710

[5] HARPER JW, 1993, CELL, V75, P805

[6] DEFECTS IN A CELL-CYCLE CHECKPOINT MAY BE RESPONSIBLE FOR THE GENOMIC INSTABILITY OF CANCER-CELLS [J]. CELL, 1992, 71 (04) : 543 - 546

[7] HARTWELL LH, 1991, GENETICS, V129, P975

[8] JENTSCH D, 1992, A REV GENET, V26, P179

[9] A MAMMALIAN-CELL CYCLE CHECKPOINT PATHWAY UTILIZING P53 AND GADD45 IS DEFECTIVE IN ATAXIA-TELANGIECTASIA [J]. CELL, 1992, 71 (04) : 587 - 597

[10] CHECKPOINTS THAT COUPLE GENE-EXPRESSION TO MORPHOGENESIS [J]. SCIENCE, 1993, 262 (5137) : 1227 - 1228